Literature DB >> 12027931

Body composition in adults with Type 1 diabetes at onset and during the first year of insulin therapy.

A M Rosenfalck1, T Almdal, J Hilsted, S Madsbad.   

Abstract

AIMS: To describe body composition in patients with Type 1 diabetes at diagnosis and during the first year after initiation of insulin therapy. RESEARCH DESIGN AND METHODS: In 10 (eight male and two female) newly onset Type 1 patients, age 31.5 +/- 3.2 years (27-37 years) (sd and range), body mass index (BMI) 20.8 +/- 1.6 (19.2-23.4) kg/m2, body composition was estimated by means of dual-energy X-ray absorptiometry (DXA) whole body scanning supplemented by estimation of total body water (TBW) (isotope dilution technique with 3H2O) at diagnosis and after 1, 3, 6 and 12 months of insulin therapy.
RESULTS: During the first year after onset of diabetes body weight (BW) increased 4.3 +/- 2.9 (0.1-8.3) kg (P = 0.0012) distributed as a 13.3% (1.6 kg) increase in total fat mass (FM) and 4.9% (2.5 kg) increase in lean body soft tissue mass (LBM). The self-reported weight loss at onset was 6.3 +/- 2.5 kg (1.5-10.0 kg). Compared with two reference populations the Metropolitan Life Insurance Co. and a healthy age and sex-matched local DXA scanned group the initial body composition data demonstrated BW 6.2 kg below ideal weight and a significant reduction of the FM (25% or -0.87 sd), whereas LBM was within the expected range.
CONCLUSIONS: During the first year after onset of Type 1 diabetes the mean increase in BW is 6.5% with a 13.3% increase in FM and a 4.9% increase in LBM. Self-reported data on premorbid BW suggest an approximately 10% reduction in BW at onset of Type 1 diabetes. Compared with a healthy reference population initial body composition data demonstrate a 25% reduction of the FM, whereas only a minor and non-significant reduction in the LBM is encountered. These data indicate that uncontrolled diabetes is rather a fat catabolic state than, as previously believed, a protein catabolic state.

Entities:  

Mesh:

Substances:

Year:  2002        PMID: 12027931     DOI: 10.1046/j.1464-5491.2002.00702.x

Source DB:  PubMed          Journal:  Diabet Med        ISSN: 0742-3071            Impact factor:   4.359


  5 in total

Review 1.  Diabetes pharmacotherapy and effects on the musculoskeletal system.

Authors:  Evangelia Kalaitzoglou; John L Fowlkes; Iuliana Popescu; Kathryn M Thrailkill
Journal:  Diabetes Metab Res Rev       Date:  2018-12-20       Impact factor: 4.876

2.  Insulin treatment normalizes retinal neuroinflammation but not markers of synapse loss in diabetic rats.

Authors:  Dustin R Masser; Heather D VanGuilder Starkey; Georgina V Bixler; Wendy Dunton; Sarah K Bronson; Willard M Freeman
Journal:  Exp Eye Res       Date:  2014-06-12       Impact factor: 3.467

3.  Impaired macrophage and satellite cell infiltration occurs in a muscle-specific fashion following injury in diabetic skeletal muscle.

Authors:  Matthew P Krause; Dhuha Al-Sajee; Donna M D'Souza; Irena A Rebalka; Jasmin Moradi; Michael C Riddell; Thomas J Hawke
Journal:  PLoS One       Date:  2013-08-12       Impact factor: 3.240

4.  Incidence of diabetes and its mortality according to body mass index in South Koreans aged 40-79 years.

Authors:  Hae Hyuk Jung; Ji In Park; Jin Seon Jeong
Journal:  Clin Epidemiol       Date:  2017-12-07       Impact factor: 4.790

5.  FoxO Transcription Factors Are Critical Regulators of Diabetes-Related Muscle Atrophy.

Authors:  Brian T O'Neill; Gourav Bhardwaj; Christie M Penniman; Megan T Krumpoch; Pablo A Suarez Beltran; Katherine Klaus; Kennedy Poro; Mengyao Li; Hui Pan; Jonathan M Dreyfuss; K Sreekumaran Nair; C Ronald Kahn
Journal:  Diabetes       Date:  2018-12-06       Impact factor: 9.461

  5 in total

北京卡尤迪生物科技股份有限公司 © 2022-2023.