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Literature DB >> 11943136

The allograft inflammatory factor-1 family of proteins.

Martin H Deininger¹, Richard Meyermann, Hermann J Schluesener.

Abstract

The allograft inflammatory factor-1 (AIF-1) is a 17 kDa interferon-gamma-inducible Ca(2+)-binding EF-hand protein that is encoded within the HLA class III genomic region. Three proteins are probably identical with AIF-1 termed Iba1 (ionized Ca(2+)-binding adapter), MRF-1 (microglia response factor) and daintain. Considerable but not complete sequence identity with AIF-1 has been described for IRT-1 (interferon-responsive transcript), BART-1 (balloon angioplasty-responsive transcript), and other, yet unassigned alternatively spliced variants. In this review, genomic and functional characteristics of AIF-1-related proteins are summarized and a common nomenclature is proposed.

Entities: Chemical Disease Gene

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Year: 2002 PMID： 11943136 DOI： 10.1016/s0014-5793(02)02430-4

Source DB: PubMed Journal: FEBS Lett ISSN： 0014-5793 Impact factor: 4.124

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Cited

25 in total

1. Resistance of Biomphalaria glabrata 13-16-R1 snails to Schistosoma mansoni PR1 is a function of haemocyte abundance and constitutive levels of specific transcripts in haemocytes.

Authors: Maureen K Larson; Randal C Bender; Christopher J Bayne
Journal: Int J Parasitol Date: 2014-03-28 Impact factor: 3.981

2. Lesion genesis in a subset of patients with multiple sclerosis: a role for innate immunity?

Authors: Christina Marik; Paul A Felts; Jan Bauer; Hans Lassmann; Kenneth J Smith
Journal: Brain Date: 2007-11 Impact factor: 13.501

3. Loss of Allograft Inflammatory Factor-1 Ameliorates Experimental Autoimmune Encephalomyelitis by Limiting Encephalitogenic CD4 T-Cell Expansion.

Authors: Prameladevi Chinnasamy; Sarah E Lutz; Dario F Riascos-Bernal; Venkatesh Jeganathan; Isabel Casimiro; Celia F Brosnan; Nicholas E S Sibinga
Journal: Mol Med Date: 2015-01-06 Impact factor: 6.354

4. Equine Arteritis Virus Has Specific Tropism for Stromal Cells and CD8⁺ T and CD21⁺ B Lymphocytes but Not for Glandular Epithelium at the Primary Site of Persistent Infection in the Stallion Reproductive Tract.

Authors: Mariano Carossino; Alan T Loynachan; Igor F Canisso; R Frank Cook; Juliana R Campos; Bora Nam; Yun Young Go; Edward L Squires; Mats H T Troedsson; Thomas Swerczek; Fabio Del Piero; Ernest Bailey; Peter J Timoney; Udeni B R Balasuriya
Journal: J Virol Date: 2017-06-09 Impact factor: 5.103

5. NFAT regulates the expression of AIF-1 and IRT-1: yin and yang splice variants of neointima formation and atherosclerosis.

Authors: Lisa M Berglund; Olga Kotova; Peter Osmark; Helena Grufman; Chen Xing; Marie-Louise Lydrup; Isabel Goncalves; Michael V Autieri; Maria F Gomez
Journal: Cardiovasc Res Date: 2011-11-23 Impact factor: 10.787

The allograft inflammatory factor-1 family of proteins.

1. Resistance of Biomphalaria glabrata 13-16-R1 snails to Schistosoma mansoni PR1 is a function of haemocyte abundance and constitutive levels of specific transcripts in haemocytes.

2. Lesion genesis in a subset of patients with multiple sclerosis: a role for innate immunity?

3. Loss of Allograft Inflammatory Factor-1 Ameliorates Experimental Autoimmune Encephalomyelitis by Limiting Encephalitogenic CD4 T-Cell Expansion.

4. Equine Arteritis Virus Has Specific Tropism for Stromal Cells and CD8⁺ T and CD21⁺ B Lymphocytes but Not for Glandular Epithelium at the Primary Site of Persistent Infection in the Stallion Reproductive Tract.

5. NFAT regulates the expression of AIF-1 and IRT-1: yin and yang splice variants of neointima formation and atherosclerosis.

6. Some Galeomorph Sharks Express a Mammalian Microglia-Specific Protein in Radial Ependymoglia of the Telencephalon.

7. Increased smooth muscle cell activation and neointima formation in response to injury in AIF-1 transgenic mice.

8. Allograft inflammatory factor-1 alleviates liver disease of BALB/c mice infected with Schistosoma japonicum.

9. AIF-1 expression regulates endothelial cell activation, signal transduction, and vasculogenesis.

10. Microglial disruption in young mice with early chronic lead exposure.