Literature DB >> 11940764

Preservation of splenic immune functions by female sex hormones after trauma-hemorrhage.

Markus W Knöferl1, Martin K Angele, Martin G Schwacha, Kirby I Bland, Irshad H Chaudry.   

Abstract

OBJECTIVES: Immune functions are markedly depressed in males but not in proestrus females after trauma-hemorrhage. Nonetheless, it is unclear what role sex steroids play in the maintenance of immune function in females after trauma-hemorrhage.
DESIGN: Prospective, controlled animal study.
SETTING: University research laboratory.
SUBJECTS: Eight-week-old female CBA/J mice.
INTERVENTIONS: Mice underwent sham-ovariectomy or ovariectomy. Two weeks thereafter, ovariectomized and proestrus sham-ovariectomized mice were subjected to laparotomy (i.e., soft tissue trauma) and hemorrhagic shock (35 +/- 5 mm Hg for 90 mins, resuscitated) or sham operation. Splenocyte proliferation and interleukin-2, interleukin-3, and interferon-gamma release were determined at 2 hrs after trauma-hemorrhage.
MEASUREMENTS AND MAIN RESULTS: These immune functional capacities were maintained in proestrus sham-ovariectomized mice after trauma-hemorrhage, whereas they were suppressed in ovariectomized mice subjected to trauma-hemorrhage. 17beta-Estradiol in vitro had no effect on splenocyte functions in proestrus sham-ovariectomized females; however, addition of 17beta-estradiol to splenocytes from ovariectomized females subjected to trauma-hemorrhage normalized immune functional capacities.
CONCLUSIONS: These findings suggest that elevated circulating 17beta-estradiol in proestrus females plays a direct role in the maintenance of immunocompetence after trauma-hemorrhage.

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Year:  2002        PMID: 11940764     DOI: 10.1097/00003246-200204000-00029

Source DB:  PubMed          Journal:  Crit Care Med        ISSN: 0090-3493            Impact factor:   7.598


  14 in total

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