Literature DB >> 1192557

Active and passive immunization against angiotensin II in the rat and rabbit. Evidence for a normal regulation of the renin-angiotensin system.

P Oster, H Bauknecht, E Hackenthal.   

Abstract

Active or passive immunization has been used repeatedly as a tool in studies on the role of the renin-angiotensin system in the control of blood pressure and kidney function. The results are not consistent among each other, and they are also at variance with other studies using different approaches. To evaluate the possible causes of these discrepancies, the biological characteristics of angiotensin antibodies were studied in rats. Following the intravenous injection of angiotensin II antibodies (purified by affinity chromatography), the plasma concentration of the antibodies declined in a two-exponential curve with half times of 11 hours and 7 days, which probably reflect distribution in the extracellular space and elimination, respectively. Plasma angiotensin II levels rose from preinjection levels of 100 pg/ml to 12,000 pg/ml within 10 minutes and then declined concomitantly with the decline in antibody concentration. We calculated that only a samll fraction of the circulating antibody was occupied by angiotensin II. Plasma renin concentrations were initially elevated both in controls and in antibody-injected rats, but they returned to the control level after 30 minutes and remained at that level throughout the rest of the experiment (10 days). This fact indicates that feedback mechanisms which control renin secretion, such as free plasma angiotensin II concentrations, are in the normal range. We therefore concluded that the renin-angiotensin system in antibody-injected rats was regulated at a normal level. Similar conditions seemed to exist in rabbits actively immunized against angiotensin II; these animals exhibited high concentrations of total immunoreactive angiotensin II (up to 200,000 pg/ml) and a small increase in plasma aldosterone and corticosterone concentrations.

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Year:  1975        PMID: 1192557     DOI: 10.1161/01.res.37.5.607

Source DB:  PubMed          Journal:  Circ Res        ISSN: 0009-7330            Impact factor:   17.367


  8 in total

Review 1.  Success and failure of vaccines against renin-angiotensin system components.

Authors:  Morris J Brown
Journal:  Nat Rev Cardiol       Date:  2009-08-25       Impact factor: 32.419

2.  Threshold pressure for the pressure-dependent renin release in the autoregulating kidney of conscious dogs.

Authors:  R Finke; R Gross; E Hackenthal; J Huber; H R Kirchheim
Journal:  Pflugers Arch       Date:  1983-10       Impact factor: 3.657

3.  A physiological role for pressure-dependent renin release in long-term blood pressure control.

Authors:  H Ehmke; P Persson; H Kirchheim
Journal:  Pflugers Arch       Date:  1987-11       Impact factor: 3.657

4.  Autoregulation of renal blood flow, glomerular filtration rate and renin release in conscious dogs.

Authors:  H R Kirchheim; H Ehmke; E Hackenthal; W Löwe; P Persson
Journal:  Pflugers Arch       Date:  1987-11       Impact factor: 3.657

5.  Chronic blockade of endogenous atrial natriuretic polypeptide (ANP) by monoclonal antibody against ANP accelerates the development of hypertension in spontaneously hypertensive and deoxycorticosterone acetate-salt-hypertensive rats.

Authors:  H Itoh; K Nakao; M Mukoyama; T Yamada; K Hosoda; G Shirakami; N Morii; A Sugawara; Y Saito; S Shiono
Journal:  J Clin Invest       Date:  1989-07       Impact factor: 14.808

6.  Baroreflex sympathetic activation increases threshold pressure for the pressure-dependent renin release in conscious dogs.

Authors:  H R Kirchheim; R Finke; E Hackenthal; W Löwe; P Persson
Journal:  Pflugers Arch       Date:  1985-09       Impact factor: 3.657

7.  Interaction between perfusion pressure and sympathetic nerves in renin release by carotid baroreflex in conscious dogs.

Authors:  R Gross; H M Hackenberg; E Hackenthal; H Kirchheim
Journal:  J Physiol       Date:  1981       Impact factor: 5.182

8.  Glutathionylation mediates angiotensin II-induced eNOS uncoupling, amplifying NADPH oxidase-dependent endothelial dysfunction.

Authors:  Keyvan Karimi Galougahi; Chia-Chi Liu; Carmine Gentile; Cindy Kok; Andrea Nunez; Alvaro Garcia; Natasha A S Fry; Michael J Davies; Clare L Hawkins; Helge H Rasmussen; Gemma A Figtree
Journal:  J Am Heart Assoc       Date:  2014-04-22       Impact factor: 5.501

  8 in total

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