Literature DB >> 11925051

Effects of tiagabine and diazepam on operant ethanol self-administration in the rat.

R Rimondini1, W Sommer, M Heilig.   

Abstract

OBJECTIVE: Benzodiazepines (BDZ) are widely used in the treatment of anxiety and ethanol withdrawal. It has been suggested that this class of compounds may increase the reinforcing value of ethanol; however, the literature is scarce. Tiagabine has recently been introduced into clinical use as an anti-epileptic drug. It acts through inhibiting gamma-aminobutyric acid (GABA) reuptake, and thus represents a pharmacodynamically novel principle for potentiating GABAergic transmission. The objective of the present study was to examine whether these two manners of modulating GABAergic transmission would affect ethanol self-administration in rats.
METHOD: Rats were trained on an operant oral ethanol self-administration task in a two-lever free-choice paradigm. When trained, subjects were treated with tiagabine (2, 6 and 18 mg/kg, intraperitoneally [i.p.]) or diazepam (0.5, 1.5 and 4.5 mg/kg, i.p.). Postsession blood alcohol concentrations and locomotor activity measures also were obtained.
RESULTS: At nonsedating doses, neither tiagabine nor diazepam affected operant ethanol self-administration. At the highest doses (18 and 4.5 mg/kg, respectively), both drugs suppressed ethanol self-administration but also induced significant suppression of locomotion, indicative of sedation.
CONCLUSIONS: Systemic administration of either the GABA-uptake blocker, tiagabine, or the GABA/BDZ agonist, diazepam, at nonsedating doses does not seem to affect oral ethanol self-administration.

Entities:  

Mesh:

Substances:

Year:  2002        PMID: 11925051

Source DB:  PubMed          Journal:  J Stud Alcohol        ISSN: 0096-882X


  8 in total

1.  Prolonged chronic ethanol exposure alters neuropeptide Y and corticotropin-releasing factor levels in the brain of adult Wistar rats.

Authors:  José R Criado; Tianmin Liu; Cindy L Ehlers; Aleksander A Mathé
Journal:  Pharmacol Biochem Behav       Date:  2011-04-17       Impact factor: 3.533

2.  Reinforcing effectiveness of midazolam, ethanol, and sucrose: behavioral economic comparison of a mixture relative to its component solutions.

Authors:  E Andrew Townsend; Donna M Platt; James K Rowlett; Peter G Roma; Kevin B Freeman
Journal:  Behav Pharmacol       Date:  2017-08       Impact factor: 2.293

3.  Ethanol, not detectably metabolized in brain, significantly reduces brain metabolism, probably via action at specific GABA(A) receptors and has measureable metabolic effects at very low concentrations.

Authors:  Caroline D Rae; Joanne E Davidson; Anthony D Maher; Benjamin D Rowlands; Mohammed A Kashem; Fatima A Nasrallah; Sundari K Rallapalli; James M Cook; Vladimir J Balcar
Journal:  J Neurochem       Date:  2013-12-18       Impact factor: 5.372

4.  GABA(A) receptor modulation during adolescence alters adult ethanol intake and preference in rats.

Authors:  Mary W Hulin; Russell J Amato; Peter J Winsauer
Journal:  Alcohol Clin Exp Res       Date:  2011-09-06       Impact factor: 3.455

5.  Zonisamide decreases ethanol intake in rats and mice.

Authors:  Clifford M Knapp; Melissa Mercado; Tara Lynn Markley; Steven Crosby; Domenic A Ciraulo; Conan Kornetsky
Journal:  Pharmacol Biochem Behav       Date:  2007-04-11       Impact factor: 3.533

6.  Effects of adolescent onset voluntary drinking followed by ethanol vapor exposure on subsequent ethanol consumption during protracted withdrawal in adult Wistar rats.

Authors:  Jose R Criado; Cindy L Ehlers
Journal:  Pharmacol Biochem Behav       Date:  2012-11-02       Impact factor: 3.533

Review 7.  The role of GABAA receptors in mediating the effects of alcohol in the central nervous system.

Authors:  Martin Davies
Journal:  J Psychiatry Neurosci       Date:  2003-07       Impact factor: 6.186

8.  Intensity and duration of chronic ethanol exposure is critical for subsequent escalation of voluntary ethanol drinking in mice.

Authors:  William C Griffin; Marcelo F Lopez; Howard C Becker
Journal:  Alcohol Clin Exp Res       Date:  2009-08-10       Impact factor: 3.455

  8 in total

北京卡尤迪生物科技股份有限公司 © 2022-2023.