Literature DB >> 11893366

Severe hypertriglyceridemia with insulin resistance is associated with systemic inflammation: reversal with bezafibrate therapy in a randomized controlled trial.

Iris J Jonkers1, Martina F Mohrschladt, Rudi G Westendorp, Arnoud van der Laarse, Augustinus H Smelt.   

Abstract

PURPOSE: To determine whether hypertriglyceridemia is associated with systemic inflammation, which may contribute to the increased cardiovascular risk in patients who have hypertriglyceridemia. In addition, we investigated whether fibrates reverse this inflammatory state. PATIENTS AND METHODS: Serum lipid levels, body mass index, insulin resistance, and inflammatory parameters were compared between 18 patients who had severe hypertriglyceridemia without cardiovascular disease and 20 normolipidemic controls. We measured the ex vivo production capacity of tumor necrosis factor (TNF)-alpha and interleukin (IL)-6 after whole-blood stimulation with lipopolysaccharide, as well as circulating levels of C-reactive protein and fibrinogen. A randomized controlled trial was conducted to determine whether bezafibrate (400 mg administered daily for 6 weeks) affected these parameters in hypertriglyceridemic patients.
RESULTS: When compared with normolipidemic controls, hypertriglyceridemic patients had significantly lower high-density lipoprotein (HDL) cholesterol and higher triglyceride levels, body mass index, and insulin resistance. In addition, hypertriglyceridemic patients had a significantly higher production capacity of TNF-alpha (mean difference, 11 700 pg/mL; 95% confidence interval [CI]: 7800 to 15,700 pg/mL]) and IL-6 (mean difference, 20,400 pg/mL; 95% CI: 7800 to 32,900 pg/mL), and higher levels of C-reactive protein (mean difference, 0.8 mg/L; 95% CI: 0.1 to 2.4 mg/L) and fibrinogen (mean difference, 0.8 g/dL; 95% CI: 0.3 to 1.3 g/dL). Bezafibrate therapy significantly increased HDL cholesterol levels, reduced triglyceride and insulin resistance levels, and reduced production capacity of TNF-alpha and IL-6, as well as levels of C-reactive protein and fibrinogen.
CONCLUSION: Systemic inflammation is present in patients who have the clinical phenotype that is associated with severe hypertriglyceridemia, and may contribute to the increased risk of cardiovascular disease in these patients. Bezafibrate has anti-inflammatory effects in these patients.

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Year:  2002        PMID: 11893366     DOI: 10.1016/s0002-9343(01)01123-8

Source DB:  PubMed          Journal:  Am J Med        ISSN: 0002-9343            Impact factor:   4.965


  44 in total

Review 1.  Fibrates for treatment of the metabolic syndrome.

Authors:  Kevin C Maki
Journal:  Curr Atheroscler Rep       Date:  2004-01       Impact factor: 5.113

2.  Weight loss induced by orlistat reverses fatty infiltration and improves hepatic fibrosis in obese patients with non-alcoholic steatohepatitis.

Authors:  Nimer Assy; Osamah Hussein; Zied Abassi
Journal:  Gut       Date:  2007-03       Impact factor: 23.059

Review 3.  Lipid therapy for cardiovascular disease with insulin resistance, diabetes, or the metabolic syndrome.

Authors:  Sander J Robins
Journal:  Curr Cardiol Rep       Date:  2005-11       Impact factor: 2.931

4.  Insulin sensitivity and mitochondrial function are improved in children with burn injury during a randomized controlled trial of fenofibrate.

Authors:  Melanie G Cree; Jennifer J Zwetsloot; David N Herndon; Ting Qian; Beatrice Morio; Ricki Fram; Arthur P Sanford; Asle Aarsland; Robert R Wolfe
Journal:  Ann Surg       Date:  2007-02       Impact factor: 12.969

5.  Orlistat reverse fatty infiltration and improves hepatic fibrosis in obese patients with nonalcoholic steatohepatitis (NASH).

Authors:  Osamah Hussein; Masha Grosovski; Sorina Schlesinger; Sergio Szvalb; Nimer Assy
Journal:  Dig Dis Sci       Date:  2007-04-03       Impact factor: 3.199

6.  Genome-wide association study indicates variants associated with insulin signaling and inflammation mediate lipoprotein responses to fenofibrate.

Authors:  Alexis C Frazier-Wood; Stella Aslibekyan; Ingrid B Borecki; Paul N Hopkins; Chao-Qiang Lai; Jose M Ordovas; Robert J Straka; Hemant K Tiwari; Donna K Arnett
Journal:  Pharmacogenet Genomics       Date:  2012-10       Impact factor: 2.089

7.  Increases in mitochondrial biogenesis impair carcinogenesis at multiple levels.

Authors:  Xiao Wang; Carlos T Moraes
Journal:  Mol Oncol       Date:  2011-08-05       Impact factor: 6.603

Review 8.  Hypertriglyceridemia and risk of coronary heart disease.

Authors:  John E Hokanson
Journal:  Curr Cardiol Rep       Date:  2002-11       Impact factor: 2.931

Review 9.  Pleiotropic effects of fibrates.

Authors:  Giulia Chinetti-Gbaguidi; Jean Charles Fruchart; Bart Staels
Journal:  Curr Atheroscler Rep       Date:  2005-09       Impact factor: 5.113

10.  Establishment of a cut-point value of serum TNF-alpha levels in the metabolic syndrome.

Authors:  Carmen M Gurrola-Díaz; Sergio Sánchez-Enriquez; Edith Oregon-Romero; Pedro M García-López; Pedro Garzón de la Mora; Blanca E Bastidas-Ramírez; Mercedes González-Hita; José F Muñoz-Valle
Journal:  J Clin Lab Anal       Date:  2009       Impact factor: 2.352

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