| Literature DB >> 11878912 |
James Arthos1, Claudia Cicala, Sara M Selig, Andrew A White, Hanumanth M Ravindranath, Donald Van Ryk, Tavis D Steenbeke, Elizabeth Machado, Prateeti Khazanie, Meredith S Hanback, Douglas B Hanback, Ronald L Rabin, Anthony S Fauci.
Abstract
We examined the role of CD4, CXCR4, and CCR5 in HIV envelope-mediated apoptosis by measuring the response of activated PBMCs to recombinant envelope proteins derived from CXCR4- and CCR5-utilizing viruses. Apoptosis of T cells was assessed by annexin-V staining and TdT-mediated dUTP-biotin nick-end labeling. Treatment of CCR5Delta32 homozygote PBMCs with a CCR5-specific envelope induced apoptosis in T cells, demonstrating that envelope--CD4 interactions are sufficient to induce apoptosis. However, a CXCR4-specific envelope induced higher levels of apoptosis than a CCR5-specific envelope, suggesting that envelope-mediated apoptosis can be enhanced by envelope--CXCR4 interactions. We conclude that envelope can induce apoptosis in T cells independently of the coreceptor specificity of a given envelope, or the expression profile of CXCR4 or CCR5 on a target cell. However, envelope--coreceptor interactions, and in particular, envelope--CXCR4 interactions, can contribute to this process.Entities:
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Year: 2002 PMID: 11878912 DOI: 10.1006/viro.2001.1266
Source DB: PubMed Journal: Virology ISSN: 0042-6822 Impact factor: 3.616