Literature DB >> 11844242

Expression of vascular endothelial growth factors and their receptors in and around intracranial arteriovenous malformations.

Toru Koizumi1, Tetsuya Shiraishi, Naoshi Hagihara, Kazuo Tabuchi, Takashi Hayashi, Teruaki Kawano.   

Abstract

OBJECTIVE: The precise mechanisms responsible for the development and growth of intracranial arteriovenous malformations (AVMs) remain unclear, but it has been hypothesized that vascular endothelial growth factors (VEGFs) might be involved in their pathogenesis. The aim of this study was to examine immunohistochemically the presence of the VEGF family (VEGF-A to -D) and their receptors (Flt-1, Flk-1, and Flt-4) in the surgically resected AVM nidus.
METHODS: The AVM nidus was surgically obtained from 31 patients with AVMs (mean age, 40.5 yr, range 13-73 yr). The mean size of the nidus was 31.6 mm (range, 15-60 mm). Formalin-fixed, paraffin-embedded specimens were stained immunohistochemically by the labeled streptavidin-biotin method with antibodies against VEGF-A to -D, as well as Flt-1, Flk-1, and Flt-4.
RESULTS: Positive staining for VEGF-A to -D was observed in the endothelial cells of the abnormal vessels involved in the AVM nidus and in the cytoplasm of astroglia surrounding it. Samples from 30 (96.8%) of 31 patients stained positive for VEGF-A, 4 (9.7%) for VEGF-B, 17 (54.5%) for VEGF-C, and 16 (51.6%) for VEGF-D. Flt-1, Flk-1, and Flt-4 were also positive chiefly, but not exclusively, in the cytoplasm of vascular endothelium and smooth muscle cells of the vascular wall. With regard to VEGF receptors, it was found that among the 31 patients studied, 19 (61.3%) were immunohistochemically positive for Flt-1, 6 (19.4%) for Flk-1, and 19 (61.3%) for Flt-4. A comparison of mean nidus size and average age at operation revealed significant differences between patients positive for VEGF-C, VEGF-D, Flt-1, or Flt-4. In contrast, there were no significant differences in nidus size and age in patients positive for VEGF-A, VEGF-B, and Flk-1.
CONCLUSION: These results strongly suggest a possible contribution of the VEGF-VEGF receptor system to the growth of intracranial AVMs.

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Year:  2002        PMID: 11844242     DOI: 10.1097/00006123-200201000-00020

Source DB:  PubMed          Journal:  Neurosurgery        ISSN: 0148-396X            Impact factor:   4.654


  25 in total

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Authors:  Brian P Walcott; Ethan A Winkler; Guy A Rouleau; Michael T Lawton
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2.  Increased expression of ephrin A1 in brain arteriovenous malformation: DNA microarray analysis.

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Journal:  Neurosurg Rev       Date:  2007-06-19       Impact factor: 3.042

Review 3.  Pathogenesis of non-hereditary brain arteriovenous malformation and therapeutic implications.

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Review 4.  Biology of cerebral arteriovenous malformations with a focus on inflammation.

Authors:  Nikolaos Mouchtouris; Pascal M Jabbour; Robert M Starke; David M Hasan; Mario Zanaty; Thana Theofanis; Dale Ding; Stavropoula I Tjoumakaris; Aaron S Dumont; George M Ghobrial; David Kung; Robert H Rosenwasser; Nohra Chalouhi
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5.  VEGF plasma levels in non-ruptured intracranial aneurysms.

Authors:  I Erol Sandalcioglu; Doreen Wende; Angelika Eggert; Jens P Regel; Dietmar Stolke; Helmut Wiedemayer
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6.  The relationship between peritumoral brain edema and the expression of vascular endothelial growth factor and its receptors in intracranial meningiomas.

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7.  Endothelial signaling and the molecular basis of arteriovenous malformation.

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8.  Evidence of endothelial progenitor cells in the human brain and spinal cord arteriovenous malformations.

Authors:  Peng Gao; Yongmei Chen; Michael T Lawton; Nicholas M Barbaro; Guo-Yuan Yang; Hua Su; Feng Ling; William L Young
Journal:  Neurosurgery       Date:  2010-10       Impact factor: 4.654

9.  Two consecutive dural arteriovenous fistulae in a child: a case report of successful treatment with gamma knife radiosurgery.

Authors:  Hung-Chuan Pan; Jason Sheehan; Chuan-Fu Huang; Dar-Yu Yang
Journal:  Childs Nerv Syst       Date:  2007-05-09       Impact factor: 1.475

10.  Nonischemic cerebral venous hypertension promotes a pro-angiogenic stage through HIF-1 downstream genes and leukocyte-derived MMP-9.

Authors:  Peng Gao; Yiqian Zhu; Feng Ling; Fanxia Shen; Brian Lee; Rodney Allanigue Gabriel; Qi Hao; Guo-Yuan Yang; Hua Su; William L Young
Journal:  J Cereb Blood Flow Metab       Date:  2009-05-27       Impact factor: 6.200

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