Literature DB >> 11822783

Estrogens ameliorate remote organ inflammation induced by burn injury in rats.

E S Ozveri1, A Bozkurt, G Haklar, S Cetinel, S Arbak, C Yeğen, B C Yeğen.   

Abstract

OBJECTIVE AND
DESIGN: The present study was designed to investigate the role of sex steroids in burn-induced remote organ injury. MATERIAL OR
SUBJECTS: Male Wistar albino rats were given burn trauma (n=39), and underwent castration or sham operation at 2 h following the burn injury. TREATMENT: Rats were injected sc with either 17beta estradiol benzoate (E2, 10 mg/kg) or an androgen receptor blocker cyproterone acetate (CPA, 25 mg/kg) or vehicle, immediately after burn and at 12 h.
METHODS: At 24 h of burn insult, rats were decapitated. Blood samples for RIA of testosterone, estradiol and tumor necrosis factor (TNF)-alpha and the tissue samples for myeloperoxidase activitiy (MPO) were taken. ANOVA student's t test was used for statistical analysis.
RESULTS: Castration, antiandrogen and E2 treatments increased plasma estradiol levels and depressed burn-induced elevation in serum TNF-alpha levels. In the liver and lung, burn-induced increase in MPO was reduced by E2 and castration, while CPA was effective in reducing neutrophil infiltration only in the liver.
CONCLUSION: We propose that treatment with estrogens or antiandrogens might be applicable in clinical situations to ameliorate systemic inflammation induced by burn.

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Year:  2001        PMID: 11822783     DOI: 10.1007/PL00000238

Source DB:  PubMed          Journal:  Inflamm Res        ISSN: 1023-3830            Impact factor:   4.575


  17 in total

Review 1.  Sex differences and estrogen modulation of the cellular immune response after injury.

Authors:  Melanie D Bird; John Karavitis; Elizabeth J Kovacs
Journal:  Cell Immunol       Date:  2008-02-21       Impact factor: 4.868

2.  The Time Course Pathological Changes After Burn Injury.

Authors:  Dan Wu; Ming Zhou; Liang Li; Jizhen Ren; Yanwei Sun; Ning Wang; Zhenyu Chen
Journal:  Inflammation       Date:  2018-10       Impact factor: 4.092

3.  Aromatase (CYP19) promoter gene polymorphism and risk of nonviral hepatitis-related hepatocellular carcinoma.

Authors:  Woon-Puay Koh; Jian-Min Yuan; Renwei Wang; Sugantha Govindarajan; Rowena Oppenheimer; Zhen Quan Zhang; Mimi C Yu; Sue Ann Ingles
Journal:  Cancer       Date:  2011-02-11       Impact factor: 6.860

Review 4.  Gammadelta T-cells: potential regulators of the post-burn inflammatory response.

Authors:  Martin G Schwacha
Journal:  Burns       Date:  2008-10-31       Impact factor: 2.744

5.  Androgen receptor-mediated inhibition of cutaneous wound healing.

Authors:  Gillian S Ashcroft; Stuart J Mills
Journal:  J Clin Invest       Date:  2002-09       Impact factor: 14.808

6.  Alpha-lipoic acid improves acetic acid-induced gastric ulcer healing in rats.

Authors:  Berna Karakoyun; Meral Yüksel; Feriha Ercan; Can Erzik; Berrak C Yeğen
Journal:  Inflammation       Date:  2009-02       Impact factor: 4.092

7.  Acute postischemic treatment with estrogen receptor-alpha agonist or estrogen receptor-beta agonist improves myocardial recovery.

Authors:  Nicholas D Vornehm; Meijing Wang; Aaron Abarbanell; Jeremy Herrmann; Brent Weil; Jiangjing Tan; Yue Wang; Megan Kelly; Daniel R Meldrum
Journal:  Surgery       Date:  2009-08       Impact factor: 3.982

8.  Estradiol treatment ameliorates acetic acid-induced gastric and colonic injuries in rats.

Authors:  Omer Günal; Berna K Oktar; Emine Ozçinar; Mustafa Sungur; Serap Arbak; Berrak Yeğen
Journal:  Inflammation       Date:  2003-12       Impact factor: 4.092

9.  Effects of estradiol and progesterone on the proinflammatory cytokine production by mononuclear cells from patients with chronic hepatitis C.

Authors:  Ying Yuan; Ichiro Shimizu; Mi Shen; Eriko Aoyagi; Hidetaka Takenaka; Tatuzo Itagaki; Mari Urata; Katsutaka Sannomiya; Nao Kohno; Katsuyoshi Tamaki; Masayuki Shono; Tetsuji Takayama
Journal:  World J Gastroenterol       Date:  2008-04-14       Impact factor: 5.742

10.  Estrogen treatment following severe burn injury reduces brain inflammation and apoptotic signaling.

Authors:  Joshua W Gatson; David L Maass; James W Simpkins; Ahamed H Idris; Joseph P Minei; Jane G Wigginton
Journal:  J Neuroinflammation       Date:  2009-10-22       Impact factor: 8.322

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