Literature DB >> 11765686

Oxidant injury in coronary heart disease (Part-I).

D P Pandya1.   

Abstract

Oxidative stress damages the heart through a series of reactions beginning with lipid peroxidation, the main process behind atherosclerosis. Antioxidant supplementation has some beneficial effects by binding with metal ions or catalysts to prevent oxidative lipid peroxidation and chain production.

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Year:  2001        PMID： 11765686     DOI： 10.1007/s12019-001-0026-0

Source DB:  PubMed          Journal:  Compr Ther        ISSN： 0098-8243


  3 in total

1.  mt-Nd2a suppresses reactive oxygen species production by mitochondrial complexes I and III.

Authors:  Aaron M Gusdon; Tatyana V Votyakova; Clayton E Mathews
Journal:  J Biol Chem       Date:  2008-02-15       Impact factor: 5.157

2.  Respiration and substrate transport rates as well as reactive oxygen species production distinguish mitochondria from brain and liver.

Authors:  Aaron M Gusdon; Gabriel A Fernandez-Bueno; Stephanie Wohlgemuth; Jenelle Fernandez; Jing Chen; Clayton E Mathews
Journal:  BMC Biochem       Date:  2015-09-10       Impact factor: 4.059

3.  Malondialdehyde-Modified LDL IgG Antibody Levels and Indices of Cardiac Function in Valvular Heart and Coronary Artery Disease Patients.

Authors:  Amir Ali Rahsepar; Asadollah Mirzaee; Fatemeh Moodi; Mohsen Moohebati; Shima Tavallaie; Fatemeh Khorashadizadeh; Behrooz Mottahedi; Jamil Esfehanizadeh; Ali Azari; Roshanak Khojasteh; Somayeh Mousavi; Roghayeh Paydar; Maral Amini; Majid Ghayour-Mobarhan; Gordon A Ferns; Amirhossein Sahebkar
Journal:  Med Princ Pract       Date:  2015-06-26       Impact factor: 1.927
  3 in total

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