Literature DB >> 11750273

Quantitative determination of dithiocarbamates in human plasma, serum, erythrocytes and urine: pharmacokinetics of broccoli sprout isothiocyanates in humans.

Lingxiang Ye1, Albena T Dinkova-Kostova, Kristina L Wade, Yuesheng Zhang, Theresa A Shapiro, Paul Talalay.   

Abstract

BACKGROUND: Humans are exposed to substantial quantities of isothiocyanates and glucosinolates from vegetables. Since dietary isothiocyanates are widely regarded as potentially important chemoprotectors against cancer, reliable methods for measuring the plasma and tissue pharmacokinetics of isothiocyanates and their dithiocarbamate metabolites are essential for defining dosing regimens.
METHODS: Isothiocyanates (ITC) and dithiocarbamates (DTC) react quantitatively with 1,2-benzenedithiol to produce 1,3-benzodithiole-2-thione that can be quantified spectroscopically. Although this cyclocondensation reaction has been highly useful for analyzing plant material and urine samples, the determination of DTC/ITC (the total quantity of DTC and ITC components in a sample that react in the cyclocondensation reaction) in blood and tissues has been hampered by their low levels and the high concentrations of proteins that interfere with the cyclocondensation reaction. The protein content of blood and tissues was reduced by the precipitation with polyethylene glycol (PEG) or ultrafiltration, and the sensitivity of the method was increased substantially by the solid phase extraction of the cyclocondensation product.
RESULTS: Pharmacokinetic measurements were made in four human volunteers who received single doses of about 200 micromol of broccoli sprout isothiocyanates (largely sulforaphane, with lesser amounts of iberin and erucin). Isothiocyanates were absorbed rapidly, reached peak concentrations of 0.943-2.27 micromol/l in plasma, serum and erythrocytes at 1 h after feeding and declined with first-order kinetics (half-life of 1.77+/-0.13 h). The cumulative excretion at 8 h was 58.3+/-2.8% of the dose. Clearance was 369+/-53 ml/min, indicating active renal tubular secretion.
CONCLUSION: A sensitive and specific method for quantifying DTC levels in human plasma, serum, and erythrocytes has been devised. Determinations of ITC/DTC levels are important because: (i) dietary isothiocyanates are of potential value in reducing the risk of cancer, and (ii) humans are extensively exposed to DTC as fungicides, insecticides, pesticides and rubber vulcanization accelerators.

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Year:  2002        PMID: 11750273     DOI: 10.1016/s0009-8981(01)00727-6

Source DB:  PubMed          Journal:  Clin Chim Acta        ISSN: 0009-8981            Impact factor:   3.786


  110 in total

Review 1.  Dietary Sulforaphane in Cancer Chemoprevention: The Role of Epigenetic Regulation and HDAC Inhibition.

Authors:  Stephanie M Tortorella; Simon G Royce; Paul V Licciardi; Tom C Karagiannis
Journal:  Antioxid Redox Signal       Date:  2014-12-19       Impact factor: 8.401

2.  Modulation of the metabolism of airborne pollutants by glucoraphanin-rich and sulforaphane-rich broccoli sprout beverages in Qidong, China.

Authors:  Thomas W Kensler; Derek Ng; Steven G Carmella; Menglan Chen; Lisa P Jacobson; Alvaro Muñoz; Patricia A Egner; Jian Guo Chen; Geng Sun Qian; Tao Yang Chen; Jed W Fahey; Paul Talalay; John D Groopman; Jian-Min Yuan; Stephen S Hecht
Journal:  Carcinogenesis       Date:  2011-11-01       Impact factor: 4.944

3.  Cruciferous vegetables, isothiocyanates, and prevention of bladder cancer.

Authors:  Omkara L Veeranki; Arup Bhattacharya; Li Tang; James R Marshall; Yuesheng Zhang
Journal:  Curr Pharmacol Rep       Date:  2015-08

Review 4.  Dietary HDAC inhibitors: time to rethink weak ligands in cancer chemoprevention?

Authors:  Roderick H Dashwood; Melinda C Myzak; Emily Ho
Journal:  Carcinogenesis       Date:  2005-11-02       Impact factor: 4.944

5.  Protection of humans by plant glucosinolates: efficiency of conversion of glucosinolates to isothiocyanates by the gastrointestinal microflora.

Authors:  Jed W Fahey; Scott L Wehage; W David Holtzclaw; Thomas W Kensler; Patricia A Egner; Theresa A Shapiro; Paul Talalay
Journal:  Cancer Prev Res (Phila)       Date:  2012-02-07

6.  The role of STAT-3 in the induction of apoptosis in pancreatic cancer cells by benzyl isothiocyanate.

Authors:  Ravi P Sahu; Sanjay K Srivastava
Journal:  J Natl Cancer Inst       Date:  2009-01-27       Impact factor: 13.506

Review 7.  Dietary agents as histone deacetylase inhibitors.

Authors:  Melinda C Myzak; Emily Ho; Roderick H Dashwood
Journal:  Mol Carcinog       Date:  2006-06       Impact factor: 4.784

Review 8.  Cruciferous Vegetables, Isothiocyanates, and Bladder Cancer Prevention.

Authors:  Besma Abbaoui; Christopher R Lucas; Ken M Riedl; Steven K Clinton; Amir Mortazavi
Journal:  Mol Nutr Food Res       Date:  2018-08-29       Impact factor: 5.914

9.  Suppression of microtubule dynamic instability and turnover in MCF7 breast cancer cells by sulforaphane.

Authors:  Olga Azarenko; Tatiana Okouneva; Keith W Singletary; Mary Ann Jordan; Leslie Wilson
Journal:  Carcinogenesis       Date:  2008-10-23       Impact factor: 4.944

10.  Direct modification of the proinflammatory cytokine macrophage migration inhibitory factor by dietary isothiocyanates.

Authors:  Kristin K Brown; Frances H Blaikie; Robin A J Smith; Joel D A Tyndall; Hongqi Lue; Jürgen Bernhagen; Christine C Winterbourn; Mark B Hampton
Journal:  J Biol Chem       Date:  2009-09-23       Impact factor: 5.157

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