Literature DB >> 11743361

Smooth muscle differentiation and cell turnover in mouse detrusor development.

N Smeulders1, A S Woolf, D T Wilcox.   

Abstract

PURPOSE: We systematically analyzed detrusor muscle differentiation in normal mice with a focus on cell turnover (proliferation and apoptosis) as well as on expression of the muscle specific proteins alpha-smooth muscle actin and desmin.
MATERIALS AND METHODS: The stages examined were embryonic days 14 and 18, and postnatal day 1 and week 6, representing a period spanning organ inception to postnatal maturity. Alpha-smooth muscle actin, desmin and proliferating cell nuclear antigen were assessed by immunohistochemical testing of whole bladders and Western blot analysis of dissected detrusor layers. Apoptosis was detected in tissue sections by end-labeling.
RESULTS: Alpha-smooth muscle actin was expressed by the detrusor layer throughout maturation with levels significantly increasing from embryonic days 14 to 18 and cytoplasmic staining gaining in uniformity postnatally. Desmin expression in the detrusor was insignificant at embryonic day 14 but increased progressively thereafter. Proliferating cell nuclear antigen expression in the detrusor was highest at organ inception and fell stepwise at each developmental stage with low levels postnatally. Apoptosis in the detrusor was only detected at embryonic day 14.
CONCLUSIONS: These results demonstrate that morphological growth of the mouse detrusor muscle is accompanied by complex serial changes in the expression of muscle specific proteins and in cell turnover. Strikingly, detrusor muscle cell differentiation and proliferation are inversely related. These detailed studies may serve as a comparison for future experiments involving aberrant mouse bladder development.

Entities:  

Mesh:

Substances:

Year:  2002        PMID: 11743361

Source DB:  PubMed          Journal:  J Urol        ISSN: 0022-5347            Impact factor:   7.450


  5 in total

1.  Urinary tract effects of HPSE2 mutations.

Authors:  Helen M Stuart; Neil A Roberts; Emma N Hilton; Edward A McKenzie; Sarah B Daly; Kristen D Hadfield; Jeffery S Rahal; Natalie J Gardiner; Simon W Tanley; Malcolm A Lewis; Emily Sites; Brad Angle; Cláudia Alves; Teresa Lourenço; Márcia Rodrigues; Angelina Calado; Marta Amado; Nancy Guerreiro; Inês Serras; Christian Beetz; Rita-Eva Varga; Mesrur Selcuk Silay; John M Darlow; Mark G Dobson; David E Barton; Manuela Hunziker; Prem Puri; Sally A Feather; Judith A Goodship; Timothy H J Goodship; Heather J Lambert; Heather J Cordell; Anand Saggar; Maria Kinali; Christian Lorenz; Kristina Moeller; Franz Schaefer; Aysun K Bayazit; Stefanie Weber; William G Newman; Adrian S Woolf
Journal:  J Am Soc Nephrol       Date:  2014-08-21       Impact factor: 10.121

2.  The Mechanism of Bladder Injury in Fetal Rats With Myelomeningocele.

Authors:  Ying Liu; Li Chen; Yunli Bi; Jian Shen; Hong Chen; Yujie Ma
Journal:  Front Neurol       Date:  2022-06-09       Impact factor: 4.086

Review 3.  Genetics of human congenital urinary bladder disease.

Authors:  Adrian S Woolf; Helen M Stuart; William G Newman
Journal:  Pediatr Nephrol       Date:  2013-04-13       Impact factor: 3.714

4.  Urinary outflow obstruction increases apoptosis and deregulates Bcl-2 and Bax expression in the fetal ovine bladder.

Authors:  Nikesh Thiruchelvam; Peter Nyirady; Donald M Peebles; Christopher H Fry; Peter M Cuckow; Adrian S Woolf
Journal:  Am J Pathol       Date:  2003-04       Impact factor: 4.307

5.  Overactivity or blockade of transforming growth factor-β each generate a specific ureter malformation.

Authors:  Filipa M Lopes; Neil A Roberts; Leo Ah Zeef; Natalie J Gardiner; Adrian S Woolf
Journal:  J Pathol       Date:  2019-10-01       Impact factor: 7.996

  5 in total

北京卡尤迪生物科技股份有限公司 © 2022-2023.