Literature DB >> 11724284

Inhibition of tumor growth by plasminogen-related protein-B.

V O Lewis1, M S O'Reilly, M Gehrmann, M Llinás, J Schaller, L Weissbach.   

Abstract

BACKGROUND: Various fragments of the fibrinolytic protein plasminogen can act as antiangiogenic factors and inhibit the growth of primary and metastatic tumors in mice. Plasminogen-related gene-B encodes a putative 9 kDa protein virtually identical to the plasminogen N-terminal activation peptide, a 77-amino acid motif that is liberated from the parent plasminogen molecule during conversion to the serine proteinase plasmin. Previous data have documented enhanced transcription of plasminogen-related gene-B in neoplastic tissues.
MATERIALS AND METHODS: We have tested the effects of recombinant versions of plasminogen-related protein-B and the plasminogen N-terminal activation peptide on the growth of tumors in mice, employing murine tumor cell lines implanted subcutaneously.
RESULTS: The recombinant plasminogen-related protein-B significantly inhibited the growth of primary tumors in mice, while recombinant plasminogen N-terminal activation peptide elicited only a slight inhibition of tumor growth.
CONCLUSION: These data suggest that plasminogen-related protein-B may have utility as a novel cancer therapeutic.

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Year:  2001        PMID: 11724284

Source DB:  PubMed          Journal:  Anticancer Res        ISSN: 0250-7005            Impact factor:   2.480


  1 in total

1.  Inhibitory effect of ginsenoside Rg3 combined with gemcitabine on angiogenesis and growth of lung cancer in mice.

Authors:  Tai-Guo Liu; Ying Huang; Dan-Dan Cui; Xiao-Bing Huang; Shu-Hua Mao; Ling-Ling Ji; Hai-Bo Song; Cheng Yi
Journal:  BMC Cancer       Date:  2009-07-23       Impact factor: 4.430

  1 in total

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