Literature DB >> 11720893

Phenotypes associated with replacement of His by Arg in the Pendred syndrome gene.

E Sato1, T Nakashima, Y Miura, A Furuhashi, A Nakayama, N Mori, H Murakami, S Naganawa, M Tadokoro.   

Abstract

BACKGROUND: Pendred syndrome is often associated with inner ear malformations, especially enlarged vestibular aqueduct (EVA). Recently, mutations in the Pendred syndrome gene (PDS) have been reported in patients with EVA, in addition to those with classical Pendred syndrome.
OBJECTIVE: The aim of this study was to investigate the genotype-phenotype correlations of PDS.
METHODS: Each of the 21 exons and flanking splice regions of PDS was analysed by direct DNA sequencing in nine patients with EVA; allele-specific amplification was performed to confirm the mutation. Genetic analyses were compared with thyroid function tests, perchlorate discharge tests, thyroid volume and pure-tone audiogram. Magnetic resonance imaging was used to determine the volume of the endolymphatic duct and sac of each patient.
RESULTS: A missense mutation, H723R, was identified in the homozygous state in three patients and in the heterozygous state in another three. Although none of the patients had goitre, increased serum thyroglobulin and an abnormal degree of iodide release were correlated with the number of mutant alleles identified. However, there was no relationship between the degree of hearing loss and the number of mutant alleles.
CONCLUSION: The present study reveals that the number of mutant alleles correlates with the degree of subclinical thyroid abnormality, but not with the degree of hearing loss in Japanese patients with the PDS missense mutation H723R.

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Year:  2001        PMID: 11720893     DOI: 10.1530/eje.0.1450697

Source DB:  PubMed          Journal:  Eur J Endocrinol        ISSN: 0804-4643            Impact factor:   6.664


  10 in total

1.  Atrophic thyroid follicles and inner ear defects reminiscent of cochlear hypothyroidism in Slc26a4-related deafness.

Authors:  Amiel A Dror; Danielle R Lenz; Shaked Shivatzki; Keren Cohen; Osnat Ashur-Fabian; Karen B Avraham
Journal:  Mamm Genome       Date:  2014-04-24       Impact factor: 2.957

2.  Origins and frequencies of SLC26A4 (PDS) mutations in east and south Asians: global implications for the epidemiology of deafness.

Authors:  H-J Park; S Shaukat; X-Z Liu; S H Hahn; S Naz; M Ghosh; H-N Kim; S-K Moon; S Abe; K Tukamoto; S Riazuddin; M Kabra; R Erdenetungalag; J Radnaabazar; S Khan; A Pandya; S-I Usami; W E Nance; E R Wilcox; S Riazuddin; A J Griffith
Journal:  J Med Genet       Date:  2003-04       Impact factor: 6.318

3.  SLC26A4 genotype, but not cochlear radiologic structure, is correlated with hearing loss in ears with an enlarged vestibular aqueduct.

Authors:  Kelly A King; Byung Yoon Choi; Christopher Zalewski; Anne C Madeo; Ani Manichaikul; Shannon P Pryor; Anne Ferruggiaro; David Eisenman; H Jeffrey Kim; John Niparko; James Thomsen; John A Butman; Andrew J Griffith; Carmen C Brewer
Journal:  Laryngoscope       Date:  2010-02       Impact factor: 3.325

4.  TSH regulates pendrin membrane abundance and enhances iodide efflux in thyroid cells.

Authors:  Liuska Pesce; Aigerim Bizhanova; Juan Carlos Caraballo; Whitney Westphal; Maria L Butti; Alejandro Comellas; Peter Kopp
Journal:  Endocrinology       Date:  2011-11-22       Impact factor: 4.736

5.  Long-term follow-up in patients with Pendred syndrome: vestibular, auditory and other phenotypes.

Authors:  Makoto Sugiura; Eisuke Sato; Tsutomu Nakashima; Junko Sugiura; Atsushi Furuhashi; Takahiko Yoshino; Atsuo Nakayama; Naoyoshi Mori; Hideki Murakami; Shinji Naganawa
Journal:  Eur Arch Otorhinolaryngol       Date:  2005-03-04       Impact factor: 2.503

6.  Mutations in the SLC26A4 (pendrin) gene in patients with sensorineural deafness and enlarged vestibular aqueduct.

Authors:  F Bogazzi; D Russo; F Raggi; F Ultimieri; S Berrettini; F Forli; L Grasso; C Ceccarelli; S Mariotti; A Pinchera; L Bartalena; E Martino
Journal:  J Endocrinol Invest       Date:  2004-05       Impact factor: 4.256

Review 7.  Minireview: The sodium-iodide symporter NIS and pendrin in iodide homeostasis of the thyroid.

Authors:  Aigerim Bizhanova; Peter Kopp
Journal:  Endocrinology       Date:  2009-02-05       Impact factor: 4.736

8.  Influence of dietary iodine deficiency on the thyroid gland in Slc26a4-null mutant mice.

Authors:  Tomoyuki Iwata; Tadao Yoshida; Masaaki Teranishi; Yoshiharu Murata; Yoshitaka Hayashi; Yasuhiko Kanou; Andrew J Griffith; Tsutomu Nakashima
Journal:  Thyroid Res       Date:  2011-06-20

9.  The iodide transporter Slc26a7 impacts thyroid function more strongly than Slc26a4 in mice.

Authors:  Naoya Yamaguchi; Atsushi Suzuki; Aya Yoshida; Tatsushi Tanaka; Kohei Aoyama; Hisashi Oishi; Yuichiro Hara; Tomoo Ogi; Izuki Amano; Satomi Kameo; Noriyuki Koibuchi; Yasuhiro Shibata; Shinya Ugawa; Haruo Mizuno; Shinji Saitoh
Journal:  Sci Rep       Date:  2022-07-04       Impact factor: 4.996

10.  Congenital goitrous hypothyroidism is caused by dysfunction of the iodide transporter SLC26A7.

Authors:  Jun Ishii; Atsushi Suzuki; Toru Kimura; Michihiro Tateyama; Tatsushi Tanaka; Takuya Yazawa; Yu Arimasu; I-Shan Chen; Kohei Aoyama; Yoshihiro Kubo; Shinji Saitoh; Haruo Mizuno; Hiroshi Kamma
Journal:  Commun Biol       Date:  2019-07-24
  10 in total

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