Literature DB >> 11641653

Phenotypic and metabolic characteristics of monocytes and granulocytes in preeclampsia.

M T Gervasi1, T Chaiworapongsa, P Pacora, N Naccasha, B H Yoon, E Maymon, R Romero.   

Abstract

OBJECTIVE: The maternal syndrome of preeclampsia has recently been attributed to a systemic intravascular inflammatory response and endothelial cell activation and dysfunction. This novel hypothesis has considerable clinical and biological implications. This study was designed to determine whether women with preeclampsia have evidence of intravascular inflammation by examination of the phenotypic and metabolic activity of granulocytes and monocytes. STUDY
DESIGN: A cross-sectional study was performed that included patients with preeclampsia (n = 31) and normal pregnancies (n = 58) matched for gestational age at blood draw. Intravascular inflammation was studied with use of flow cytometry. Peripheral venous blood was assayed to determine granulocyte and monocyte phenotype with the use of monoclonal antibodies for selective cluster differentiation (CD) antigens. The panel of antibodies included CD11b, CD14, CD16, CD18, CD49d, CD62L, CD64, CD66b, and HLA-DR. The quantity of basal intracellular reactive oxygen species and oxidative burst was assessed. Results were reported as mean channel brightness or intensity of detected fluorescence. Analysis was conducted with nonparametric statistics. A P value <.01 was considered to be significant.
RESULTS: Preeclampsia was associated with a significant increase in mean channel brightness for CD11b on granulocytes and monocytes but lower mean channel brightness for CD62L on granulocytes than those from women with normal pregnancy (P <.01 for each). Basal intracellular reactive oxygen species were increased in monocytes but not in granulocytes. The oxidative burst was higher in both cell types.
CONCLUSION: Preeclampsia is associated with phenotypic and metabolic changes in granulocytes and monocytes.

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Year:  2001        PMID: 11641653     DOI: 10.1067/mob.2001.117311

Source DB:  PubMed          Journal:  Am J Obstet Gynecol        ISSN: 0002-9378            Impact factor:   8.661


  78 in total

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5.  Interleukin-33 in the human placenta.

Authors:  Vanessa Topping; Roberto Romero; Nandor Gabor Than; Adi L Tarca; Zhonghui Xu; Sun Young Kim; Bing Wang; Lami Yeo; Chong Jai Kim; Sonia S Hassan; Jung-Sun Kim
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6.  Long-term alterations in maternal plasma proteome after sFlt1-induced preeclampsia in mice.

Authors:  Egle Bytautiene; Nataliya Bulayeva; Geeta Bhat; Li Li; Kevin P Rosenblatt; George R Saade
Journal:  Am J Obstet Gynecol       Date:  2013-03-13       Impact factor: 8.661

7.  Preeclampsia and pregnancies with small-for-gestational age neonates have different profiles of complement split products.

Authors:  Eleazar Soto; Roberto Romero; Karina Richani; Jimmy Espinoza; Tinnakorn Chaiworapongsa; Jyh Kae Nien; Sam S Edwin; Yeon Mee Kim; Joon Seok Hong; Luis F Goncalves; Lami Yeo; Moshe Mazor; Sonia S Hassan; Juan Pedro Kusanovic
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8.  Could alterations in maternal plasma visfatin concentration participate in the phenotype definition of preeclampsia and SGA?

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Journal:  J Matern Fetal Neonatal Med       Date:  2010-08

9.  Increased Neutrophil Activation and Plasma DNA Levels in Patients with Pre-Eclampsia.

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Review 10.  Oxidative stress in the placenta.

Authors:  Leslie Myatt; Xiaolan Cui
Journal:  Histochem Cell Biol       Date:  2004-07-10       Impact factor: 4.304

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