Literature DB >> 11606501

Macrophage-derived IL-18-mediated intestinal inflammation in the murine model of Crohn's disease.

T Kanai1, M Watanabe, A Okazawa, T Sato, M Yamazaki, S Okamoto, H Ishii, T Totsuka, R Iiyama, R Okamoto, M Ikeda, M Kurimoto, K Takeda, S Akira, T Hibi.   

Abstract

BACKGROUND & AIMS: Crohn's disease (CD) is associated with an increased number of infiltrating macrophages, which release a variety of proinflammatory cytokines. Interleukin (IL)-18 has been implicated in the modulation of mucosal CD4(+) T cells towards Th1 responses, which are implicated in the pathogenesis of CD. Here we assess the role of macrophages and of IL-18 in the murine model of intestinal inflammation that mimics the immunologic characteristics of human CD.
METHODS: Colitis was induced in C57BL/6 mice immunized with 2,4,6-trinitrobenzene sulfonic acid (TNBS) followed by rectal administration of TNBS in ethanol. Mice were treated with either an antibody directed against macrophages conjugated to the ribosome-inactivating protein saporin (anti-Mac-1-saporin) or with a neutralizing antibody against IL-18. In addition, we assessed whether an identical TNBS immunization/challenge protocol could induce colitis in IL-18(-/-) mice.
RESULTS: The colonic mucosa of TNBS-treated mice was marked by infiltration of Mac-1-positive macrophages and up-regulation of IL-18. The administration of the anti-Mac-1-saporin antibody or the neutralizing anti-IL-18 antibody resulted in a dramatic attenuation of mucosal inflammation in this model. In addition, TNBS was unable to induce significant colitis in the IL-18(-/-) mice.
CONCLUSIONS: Our data underscore the pivotal role of macrophages, and the macrophage-derived IL-18, in the establishment of TNBS-induced colitis in mice. Our results highlight the potential use of therapy directed against IL-18 in the treatment of patients with CD.

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Year:  2001        PMID: 11606501     DOI: 10.1053/gast.2001.28021

Source DB:  PubMed          Journal:  Gastroenterology        ISSN: 0016-5085            Impact factor:   22.682


  56 in total

Review 1.  Role of the innate immune system in the development of chronic colitis.

Authors:  Takanori Kanai; Ryoichi Ilyama; Takahiro Ishikura; Koji Uraushihara; Teruji Totsuka; Motomi Yamazaki; Tetsuya Nakamuma; Mamoru Watanabe
Journal:  J Gastroenterol       Date:  2002-11       Impact factor: 7.527

2.  Epithelial IL-18 Equilibrium Controls Barrier Function in Colitis.

Authors:  Roni Nowarski; Ruaidhrí Jackson; Nicola Gagliani; Marcel R de Zoete; Noah W Palm; Will Bailis; Jun Siong Low; Christian C D Harman; Morven Graham; Eran Elinav; Richard A Flavell
Journal:  Cell       Date:  2015-12-03       Impact factor: 41.582

3.  Treatment with interleukin-18 binding protein ameliorates Toxoplasma gondii-induced small intestinal pathology that is induced by bone marrow cell-derived interleukin-18.

Authors:  D Struck; I Frank; S Enders; U Steinhoff; C Schmidt; A Stallmach; O Liesenfeld; M M Heimesaat
Journal:  Eur J Microbiol Immunol (Bp)       Date:  2012-09-10

4.  Novel anti-inflammatory agent 3-[(dodecylthiocarbonyl)-methyl]-glutarimide ameliorates murine models of inflammatory bowel disease.

Authors:  Nobuki Ichikawa; Kenichiro Yamashita; Tohru Funakoshi; Shin Ichihara; Moto Fukai; Masaomi Ogura; Nozomi Kobayashi; Masaaki Zaitsu; Tadashi Yoshida; Susumu Shibasaki; Yasuyuki Koshizuka; Yusuke Tsunetoshi; Masanori Sato; Takahiro Einama; Michitaka Ozaki; Kazuo Umezawa; Tomomi Suzuki; Satoru Todo
Journal:  Inflamm Res       Date:  2015-12-18       Impact factor: 4.575

5.  TGF-β2 suppresses macrophage cytokine production and mucosal inflammatory responses in the developing intestine.

Authors:  Akhil Maheshwari; David R Kelly; Teodora Nicola; Namasivayam Ambalavanan; Sunil K Jain; Joanne Murphy-Ullrich; Mohammad Athar; Masako Shimamura; Vineet Bhandari; Charles Aprahamian; Reed A Dimmitt; Rosa Serra; Robin K Ohls
Journal:  Gastroenterology       Date:  2010-09-24       Impact factor: 22.682

6.  Possible involvement of muscularis resident macrophages in impairment of interstitial cells of Cajal and myenteric nerve systems in rat models of TNBS-induced colitis.

Authors:  Kazuya Kinoshita; Kazuhide Horiguchi; Masahiko Fujisawa; Fuyu Kobirumaki; Shigeru Yamato; Masatoshi Hori; Hiroshi Ozaki
Journal:  Histochem Cell Biol       Date:  2006-07-27       Impact factor: 4.304

7.  Increased expression of Toll-like receptor (TLR) 2 and TLR4 in the colonic mucosa of children with inflammatory bowel disease.

Authors:  B Szebeni; G Veres; A Dezsõfi; K Rusai; A Vannay; M Mraz; E Majorova; A Arató
Journal:  Clin Exp Immunol       Date:  2007-11-07       Impact factor: 4.330

8.  Human intestinal epithelial cell-derived interleukin (IL)-18, along with IL-2, IL-7 and IL-15, is a potent synergistic factor for the proliferation of intraepithelial lymphocytes.

Authors:  A Okazawa; T Kanai; K Nakamaru; T Sato; N Inoue; H Ogata; Y Iwao; M Ikeda; T Kawamura; S Makita; K Uraushihara; R Okamoto; M Yamazaki; M Kurimoto; H Ishii; M Watanabe; T Hibi
Journal:  Clin Exp Immunol       Date:  2004-05       Impact factor: 4.330

Review 9.  Role of cytokines in inflammatory bowel disease.

Authors:  Fausto Sanchez-Munoz; Aaron Dominguez-Lopez; Jesus-K Yamamoto-Furusho
Journal:  World J Gastroenterol       Date:  2008-07-21       Impact factor: 5.742

10.  Neurotensin induces IL-6 secretion in mouse preadipocytes and adipose tissues during 2,4,6,-trinitrobenzensulphonic acid-induced colitis.

Authors:  Hon-Wai Koon; You Sun Kim; Hua Xu; Aatish Kumar; Dezheng Zhao; Iordanes Karagiannides; Paul R Dobner; Charalabos Pothoulakis
Journal:  Proc Natl Acad Sci U S A       Date:  2009-05-14       Impact factor: 11.205

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