Literature DB >> 11591838

Poststroke dementia: incidence and relationship to prestroke cognitive decline.

H Hénon1, I Durieu, D Guerouaou, F Lebert, F Pasquier, D Leys.   

Abstract

OBJECTIVE: To evaluate the 3-year incidence of poststroke dementia (PSD) and the influence of prestroke cognitive decline.
METHODS: The authors evaluated prestroke cognitive functions in 202 consecutive stroke patients > or =40 years old using the Informant Questionnaire on Cognitive Decline in the Elderly (IQCODE), with a cut-off of 104 for the diagnosis of dementia. Six months and then annually after stroke, dementia was reassessed. The diagnosis of dementia was based on the International Classification of Diseases, 10th revision criteria in survivors who underwent a visit with a neurologist, or on the IQCODE score obtained by telephone contact with the family in survivors who did not. Statistics were performed using life-table methods.
RESULTS: Thirty-three patients were excluded because of prestroke dementia. In the 169 remaining patients, the cumulative proportion of patients with dementia was 28.5% at the end of the follow-up period, with most of PSD occurring during the first 6 months. Using multivariate analysis, independent predictors of PSD were aging, preexisting cognitive decline, severity of deficit at admission, diabetes mellitus, and silent infarcts. Leukoaraiosis was an independent predictor of PSD when prestroke cognitive decline was not taken into account. The presumed etiology of dementia was vascular dementia (VaD) in two-thirds of patients and AD in one-third.
CONCLUSIONS: The risk of PSD is high, and increased in patients with prestroke cognitive decline, with about one-third of patients meeting the criteria for AD and two-thirds meeting the criteria for VaD. These results confirm that, in stroke patients, an underlying degenerative pathology may play a role in the development of PSD.

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Year:  2001        PMID: 11591838     DOI: 10.1212/wnl.57.7.1216

Source DB:  PubMed          Journal:  Neurology        ISSN: 0028-3878            Impact factor:   9.910


  63 in total

1.  Diagnosis, risk factors, and treatment of vascular dementia.

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2.  Cerebrovascular disease, β-amyloid, and cognition in aging.

Authors:  Natalie L Marchant; Bruce R Reed; Charles S DeCarli; Cindee M Madison; Michael W Weiner; Helena C Chui; William J Jagust
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3.  Changes in memory before and after stroke differ by age and sex, but not by race.

Authors:  Qianyi Wang; Iván Mejía-Guevara; Pamela M Rist; Stefan Walter; Benjamin D Capistrant; M Maria Glymour
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4.  Delirium as a predictor of long-term cognitive impairment in survivors of critical illness.

Authors:  Timothy D Girard; James C Jackson; Pratik P Pandharipande; Brenda T Pun; Jennifer L Thompson; Ayumi K Shintani; Sharon M Gordon; Angelo E Canonico; Robert S Dittus; Gordon R Bernard; E Wesley Ely
Journal:  Crit Care Med       Date:  2010-07       Impact factor: 7.598

Review 5.  A review of screening tests for cognitive impairment.

Authors:  Breda Cullen; Brian O'Neill; Jonathan J Evans; Robert F Coen; Brian A Lawlor
Journal:  J Neurol Neurosurg Psychiatry       Date:  2006-12-18       Impact factor: 10.154

6.  Influence of pre-existing dementia on the risk of post-stroke epileptic seizures.

Authors:  C Cordonnier; H Hénon; P Derambure; F Pasquier; D Leys
Journal:  J Neurol Neurosurg Psychiatry       Date:  2005-12       Impact factor: 10.154

7.  Demographic and CT scan features related to cognitive impairment in the first year after stroke.

Authors:  S M C Rasquin; F R J Verhey; R J van Oostenbrugge; R Lousberg; J Lodder
Journal:  J Neurol Neurosurg Psychiatry       Date:  2004-11       Impact factor: 10.154

8.  Biological and imaging predictors of cognitive impairment after stroke: a systematic review.

Authors:  Barbara Casolla; François Caparros; Charlotte Cordonnier; Stéphanie Bombois; Hilde Hénon; Régis Bordet; Francesco Orzi; Didier Leys
Journal:  J Neurol       Date:  2018-10-22       Impact factor: 4.849

9.  Modifications of tau protein after cerebral ischemia and reperfusion in rats are similar to those occurring in Alzheimer's disease - Hyperphosphorylation and cleavage of 4- and 3-repeat tau.

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Review 10.  Epidemiology of Alzheimer disease.

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