Literature DB >> 11587398

Diverticular disease and treatment with gastric acid inhibitors do not predispose to peritonitis of enteric origin in peritoneal dialysis patients.

G del Peso1, M A Bajo, L Gadola, I Millán, R Codoceo, O Celadilla, M J Castro, A Aguilera, F Gil, R Selgas.   

Abstract

OBJECTIVE: Enteric peritonitis (EP) is an infrequent complication of peritoneal dialysis (PD), with severe consequences for peritoneal membrane viability and patient outcome. Factors such as diverticular disease and gastric acid inhibitors have been implicated in its appearance. We investigated several risk factors, including those mentioned below, that can influence the development of EP.
DESIGN: Retrospective cross-sectional study.
SETTING: Tertiary-care public university hospital. PATIENTS: Fifty-seven PD patients treated in our PD unit during August 1998. MAIN OUTCOME MEASURES: A barium enema was performed on 50 of the 57 patients (the remaining 7 patients refused it) in order to exclude the presence of diverticulosis. All episodes of peritonitis occurring in those patients, including EP, were registered. Enteric peritonitis was defined as that caused by gram-positive, gram-negative, or fungus micro-organisms that colonized the intestinal tract, excluding episodes secondary to genitourinary tract or peritoneal catheter exit-site infections.
RESULTS: Twenty-four patients showed diverticular disease in the barium enema, but only 5 of them (21%) had any EP episode. Five of the 26 patients with no diverticula (19%) had EP. Fifty-five episodes of peritonitis were reported in 21 patients; 15 episodes of EP (27.3% of all) developed in 11 patients. Seven of the 11 patients (64%) required peritoneal catheter removal and 3 of them (27%) finally were transferred to hemodialysis due to consequences of the EP episode. Logistic regression analysis did not find any of the independent variables analyzed (age, sex, time on PD, type of PD, peritoneal transport parameters, presence of polycystic kidney disease, constipation or diverticulosis, or treatment with gastric acid inhibitors, or phosphate-binding agents) to be risk factors for developing EP.
CONCLUSIONS: Neither diverticulosis nor treatment with gastric acid inhibitors seem to be risk factors for developing peritonitis of enteric origin in PD patients. This type of peritonitis has to be promptly identified and treated in order to diminish the high frequency of peritoneal catheter removal and PD dropout due to such episodes.

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Year:  2001        PMID: 11587398

Source DB:  PubMed          Journal:  Perit Dial Int        ISSN: 0896-8608            Impact factor:   1.756


  4 in total

1.  Impact of age on peritonitis risk in peritoneal dialysis patients: an era effect.

Authors:  Sharon J Nessim; Joanne M Bargman; Peter C Austin; Ken Story; Sarbjit V Jassal
Journal:  Clin J Am Soc Nephrol       Date:  2008-11-05       Impact factor: 8.237

2.  Effect of gastric acid suppressants and prokinetics on peritoneal dialysis-related peritonitis.

Authors:  Ji Eun Kwon; Seong-Joon Koh; Jaeyoung Chun; Ji Won Kim; Byeong Gwan Kim; Kook Lae Lee; Jong Pil Im; Joo Sung Kim; Hyun Chae Jung
Journal:  World J Gastroenterol       Date:  2014-07-07       Impact factor: 5.742

Review 3.  The Gut in Older Patients on Peritoneal Dialysis.

Authors:  Stella Setyapranata; Stephen G Holt
Journal:  Perit Dial Int       Date:  2015-11       Impact factor: 1.756

4.  Inhibition of Gastric Acid Secretion by H2 Receptor Antagonists Associates a Definite Risk of Enteric Peritonitis and Infectious Mortality in Patients Treated with Peritoneal Dialysis.

Authors:  Miguel Pérez-Fontan; Daniela Machado Lopes; Alba García Enríquez; Beatriz López-Calviño; Andrés López-Muñiz; Teresa García Falcón; Ana Rodríguez-Carmona
Journal:  PLoS One       Date:  2016-02-12       Impact factor: 3.240

  4 in total

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