Literature DB >> 11549905

Identification of T helper cell subsets in continuous ambulatory peritoneal dialysis patients.

T Yokoyama1, K Nitta, K Futatsuyama, T Hayashi, K Honda, K Uchida, A Kawashima, W Yumura, H Nihei.   

Abstract

BACKGROUND/AIM: End-stage renal failure induces a clinical state of immunodeficiency with a higher incidence of infections and a higher mortality due to infectious complications as compared with the normal population. A crucial element in this immune response is the development of a network of cytokines. CD4-positive T helper (Th) cells differentiate into either Th1 or Th2 cells in response to various stimuli. It is important to examine whether the impaired immune responses in patients with end-stage renal disease are due to an enhanced Th1 response or a reduced number of Th2 cells.
METHOD: Using a newly developed immunofluorescence staining of intracellular cytokines for flow cytometric analysis, we studied Th subsets in 24 patients on continuous ambulatory peritoneal dialysis (CAPD).
RESULTS: The proportions of Th1 cells did not differ between CAPD patients (18.7 +/- 1.8%) and controls (19.6 +/- 1.5%). Unlike Th1 cells, a significantly higher proportion of CD4 cells in CAPD patients is characterized by a Th2-type cytokine secretion pattern as compared with healthy controls: 2.9 +/- 0.8% versus 1.7 +/- 0.4%.
CONCLUSION: These data demonstrate that Th cells from CAPD patients manifest a dysregulated differentiation profile characterized by a major increase in the percentage of Th2 cells and by a normal percentage of Th1 cells. Copyright 2001 S. Karger AG, Basel

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Year:  2001        PMID: 11549905     DOI: 10.1159/000046070

Source DB:  PubMed          Journal:  Nephron        ISSN: 1660-8151            Impact factor:   2.847


  5 in total

Review 1.  T Helper 17/Regulatory T Cell Balance and Experimental Models of Peritoneal Dialysis-Induced Damage.

Authors:  Georgios Liappas; Guadalupe Tirma Gónzalez-Mateo; Pedro Majano; José Antonio Sánchez-Tomero; Marta Ruiz-Ortega; Raquel Rodrigues Díez; Pilar Martín; Raquel Sanchez-Díaz; Rafael Selgas; Manuel López-Cabrera; Abelardo Aguilera Peralta
Journal:  Biomed Res Int       Date:  2015-05-03       Impact factor: 3.411

2.  Paricalcitol reduces peritoneal fibrosis in mice through the activation of regulatory T cells and reduction in IL-17 production.

Authors:  Guadalupe T González-Mateo; Vanessa Fernández-Míllara; Teresa Bellón; Georgios Liappas; Marta Ruiz-Ortega; Manuel López-Cabrera; Rafael Selgas; Luiz S Aroeira
Journal:  PLoS One       Date:  2014-10-03       Impact factor: 3.240

3.  Th2-biased GATA-3 transgenic mice developed severe experimental peritoneal fibrosis compared with Th1-biased T-bet and Th17-biased RORγt transgenic mice.

Authors:  Keigyou Yoh; Masami Ojima; Satoru Takahashi
Journal:  Exp Anim       Date:  2015-07-06

4.  Mechanisms of Peritoneal Fibrosis: Focus on Immune Cells-Peritoneal Stroma Interactions.

Authors:  Michela Terri; Flavia Trionfetti; Claudia Montaldo; Marco Cordani; Marco Tripodi; Manuel Lopez-Cabrera; Raffaele Strippoli
Journal:  Front Immunol       Date:  2021-03-29       Impact factor: 7.561

5.  The Prevalence of Toxoplasma gondii in Hemodialysis Patients.

Authors:  Masomeh Bayani; Amrollah Mostafazadeh; Farsheed Oliaee; Narges Kalantari
Journal:  Iran Red Crescent Med J       Date:  2013-10-05       Impact factor: 0.611

  5 in total

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