Literature DB >> 11547303

Vascular cell adhesion molecule-1 up-regulation and phenotypic modulation of vascular smooth muscle cells predate mononuclear infiltration in transplant arteriopathy.

S Sai1, A Iwata, R Thomas, M D Allen.   

Abstract

OBJECTIVE: Expression of embryonic myosin heavy chain isoforms and vascular cell adhesion molecule-1 by neointimal vascular smooth muscle cells are independent indicators of atherosclerotic plaque development in both human beings and experimental animal models. We examined the chronologic change in smooth muscle cell myosin heavy chain isoforms, vascular cell adhesion molecule-1 expression, and mononuclear cell infiltration in a carotid arterial transplant model to ascertain whether similar phenotypic changes would occur in transplant arteriopathy.
METHODS: Transplanted rabbit carotid arteries were examined at 7, 14, 21, and 35 days (n = 5, 7, 6, and 5, respectively). Lesion progression and the prevalence of smooth muscle cell myosin heavy chain isoforms, T-lymphocytes, macrophages, and vascular cell adhesion molecule-1 expression were evaluated immunohistochemically by computerized image analysis.
RESULTS: In this carotid arterial transplant model, the intima/media area ratio increased significantly by 35 days (P =.01) as cell density decreased (P =.01), suggesting extracellular matrix elaboration. Intimal smooth muscle cells expressing embryonic phenotypes were seen as early as 7 days, a phenotypic change that predated mononuclear cell infiltration of the graft by at least 7 days. By 35 days, up to 70% of intimal smooth muscle cells expressed the embryonic phenotype, coinciding with the transition from inflammatory to chronic lesions. Although, in early lesions, vascular cell adhesion molecule-1 was identified on luminal endothelium overlying mononuclear infiltrates, in advanced lesions vascular cell adhesion molecule-1 was identified primarily on intimal vascular smooth muscle cells.
CONCLUSIONS: Overall, these vascular smooth muscle cell changes mark important early events in transplant arteriopathy that may not be ameliorated by immunosuppressive regimens in routine use.

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Year:  2001        PMID: 11547303     DOI: 10.1067/mtc.2001.113601

Source DB:  PubMed          Journal:  J Thorac Cardiovasc Surg        ISSN: 0022-5223            Impact factor:   5.209


  4 in total

1.  A20-mediated modulation of inflammatory and immune responses in aortic allografts and development of transplant arteriosclerosis.

Authors:  Jeffrey J Siracuse; Mark D Fisher; Cleide G da Silva; Clayton R Peterson; Eva Csizmadia; Herwig P Moll; Scott M Damrauer; Peter Studer; Lynn Y Choi; Sanah Essayagh; Elzbieta Kaczmarek; Elizabeth R Maccariello; Andy Lee; Soizic Daniel; Christiane Ferran
Journal:  Transplantation       Date:  2012-02-27       Impact factor: 4.939

2.  A new carotid artery transplantation model of rats.

Authors:  Sihai Gao; Ping Li; Jinping Zhao; Yunfeng Zhang; Yu Sun
Journal:  J Huazhong Univ Sci Technolog Med Sci       Date:  2009-02-18

3.  A20 Haploinsufficiency Aggravates Transplant Arteriosclerosis in Mouse Vascular Allografts: Implications for Clinical Transplantation.

Authors:  Herwig P Moll; Andy Lee; Clayton R Peterson; Jesus Revuelta Cervantes; Brandon M Wojcik; Anshul Parulkar; Alessandra Mele; Philip J LoGerfo; Jeffrey J Siracuse; Eva Csizmadia; Cleide G da Silva; Christiane Ferran
Journal:  Transplantation       Date:  2016-11       Impact factor: 4.939

4.  Chemokine-like factor 1 (CKLF1) aggravates neointimal hyperplasia through activating the NF-κB /VCAM-1 pathway.

Authors:  Xinnong Liu; Chengjia Qu; Yongbao Zhang; Jie Fang; Lequn Teng; Rujiao Zhang; Xiangyu Zhang; Chenyang Shen
Journal:  FEBS Open Bio       Date:  2020-08-14       Impact factor: 2.693

  4 in total

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