Literature DB >> 11427448

Accumulation of free 3-hydroxy fatty acids in the culture media of fibroblasts from patients deficient in long-chain l-3-hydroxyacyl-CoA dehydrogenase: a useful diagnostic aid.

P M Jones1, M Moffitt, D Joseph, P A Harthcock, R L Boriack, J A Ibdah, A W Strauss, M J Bennett.   

Abstract

BACKGROUND: The diagnosis of long-chain L-3-hydroxy-acyl-coenzyme A dehydrogenase (LCHAD) deficiency frequently requires the study of cultured fibroblasts. We developed such a test that does not require disruption and loss of the cells.
METHODS: We measured free 3-hydroxy fatty acids (3-OHFAs) in media of skin fibroblasts cultures from 11 patients with a genetic deficiency of LCHAD and the associated disorder of mitochondrial trifunctional protein (MTFP). Fibroblasts were cultured for 24 h with 100 micromol/L nonisotopic palmitate added. 3-OHFAs were measured by selected-ion monitoring, stable-isotope dilution gas chromatography-mass spectrometry with [(13)C]-labeled internal standards.
RESULTS: 3-OH-hexadecanoic and 3-OH-tetradecanoic FAs were increased 14- and 11-fold, respectively, in all patients with LCHAD or MTFP deficiency when compared with control fibroblast cell lines after overnight incubation with palmitate. 3-OH-dodecanoic FA demonstrated a modest, fivefold increase in LCHAD-deficient cells. The concentrations of all 3-OHFAs were similar whether or not the medium samples were hydrolyzed to release conjugated species such as acylcarnitines, suggesting that 3-OHFAs accumulate in the media as free FAs.
CONCLUSIONS: Measurement of 3-OHFA excretion from LCHAD- or MTFP-deficient cell lines can be used as a diagnostic tool. Free FAs are the predominant form of these abnormal metabolic intermediates in culture media.

Entities:  

Mesh:

Substances:

Year:  2001        PMID: 11427448

Source DB:  PubMed          Journal:  Clin Chem        ISSN: 0009-9147            Impact factor:   8.327


  6 in total

1.  Effect of optimal dietary therapy upon visual function in children with long-chain 3-hydroxyacyl CoA dehydrogenase and trifunctional protein deficiency.

Authors:  Melanie B Gillingham; Richard G Weleber; Martha Neuringer; William E Connor; Monte Mills; Sandy van Calcar; James Ver Hoeve; Jon Wolff; Cary O Harding
Journal:  Mol Genet Metab       Date:  2005-07-22       Impact factor: 4.797

2.  Long-chain 3-hydroxy fatty acids accumulating in long-chain 3-hydroxyacyl-CoA dehydrogenase and mitochondrial trifunctional protein deficiencies uncouple oxidative phosphorylation in heart mitochondria.

Authors:  Anelise M Tonin; Alexandre U Amaral; Estela N B Busanello; Mateus Grings; Roger F Castilho; Moacir Wajner
Journal:  J Bioenerg Biomembr       Date:  2012-10-13       Impact factor: 2.945

Review 3.  Mitochondrial trifunctional protein defects: clinical implications and therapeutic approaches.

Authors:  R Scott Rector; R Mark Payne; Jamal A Ibdah
Journal:  Adv Drug Deliv Rev       Date:  2008-07-04       Impact factor: 15.470

Review 4.  Role of 3-Hydroxy Fatty Acid-Induced Hepatic Lipotoxicity in Acute Fatty Liver of Pregnancy.

Authors:  Sathish Kumar Natarajan; Jamal A Ibdah
Journal:  Int J Mol Sci       Date:  2018-01-22       Impact factor: 5.923

5.  TFPa/HADHA is required for fatty acid beta-oxidation and cardiolipin re-modeling in human cardiomyocytes.

Authors:  Jason W Miklas; Elisa Clark; Shiri Levy; Damien Detraux; Andrea Leonard; Kevin Beussman; Megan R Showalter; Alec T Smith; Peter Hofsteen; Xiulan Yang; Jesse Macadangdang; Tuula Manninen; Daniel Raftery; Anup Madan; Anu Suomalainen; Deok-Ho Kim; Charles E Murry; Oliver Fiehn; Nathan J Sniadecki; Yuliang Wang; Hannele Ruohola-Baker
Journal:  Nat Commun       Date:  2019-10-11       Impact factor: 14.919

Review 6.  Recent Advances in the Pathophysiology of Fatty Acid Oxidation Defects: Secondary Alterations of Bioenergetics and Mitochondrial Calcium Homeostasis Caused by the Accumulating Fatty Acids.

Authors:  Alexandre Umpierrez Amaral; Moacir Wajner
Journal:  Front Genet       Date:  2020-11-27       Impact factor: 4.599

  6 in total

北京卡尤迪生物科技股份有限公司 © 2022-2023.