Literature DB >> 11375962

Serine protease inhibitor causes F-actin redistribution and inhibition of calcium-mediated secretion in pancreatic acini.

V P Singh1, A K Saluja, L Bhagat, A J Hietaranta, A Song, A Mykoniatis, G J Van Acker, M L Steer.   

Abstract

BACKGROUND & AIMS: The present study was undertaken to evaluate the role of serine proteases in regulating digestive enzyme secretion in pancreatic acinar cells.
METHODS: Isolated acini were stimulated by various secretagogues in the presence or absence of cell-permeant serine protease inhibitors 4-(2-aminoethyl)-benzenesulfonyl fluoride and N(alpha)-p-tosyl-L-phenylalanine chloromethyl ketone. F-actin distribution was studied after staining with rhodamine phalloidin.
RESULTS: Both cell-permeant serine protease inhibitors blocked amylase secretion in response to secretagogues that use calcium as a second messenger (e.g., cerulein, carbamylcholine, and bombesin) but not to those that use adenosine 3',5'-cyclic monophosphate (cAMP) as a second messenger (e.g., secretin and vasoactive intestinal polypeptide). Incubation of the acini with these inhibitors also resulted in a dramatic redistribution of the F-actin cytoskeleton. This redistribution was energy dependent. Similar redistribution of F-actin from the apical to the basolateral region was also observed when acini were incubated with a supramaximally stimulating concentration of cerulein, which is known to inhibit secretion.
CONCLUSIONS: These results suggest that a serine protease activity is essential for maintaining the normal apical F-actin distribution; its inhibition redistributes F-actin from the apical to the basolateral region and blocks secretion induced by secretagogues that act via calcium. cAMP reverses the F-actin redistribution and hence cAMP-mediated secretion is not affected.

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Year:  2001        PMID: 11375962     DOI: 10.1053/gast.2001.24883

Source DB:  PubMed          Journal:  Gastroenterology        ISSN: 0016-5085            Impact factor:   22.682


  13 in total

1.  Phosphatidylinositol 3-kinase-dependent activation of trypsinogen modulates the severity of acute pancreatitis.

Authors:  V P Singh; A K Saluja; L Bhagat; G J van Acker; A M Song; S P Soltoff; L C Cantley; M L Steer
Journal:  J Clin Invest       Date:  2001-11       Impact factor: 14.808

2.  Lipotoxicity causes multisystem organ failure and exacerbates acute pancreatitis in obesity.

Authors:  Sarah Navina; Chathur Acharya; James P DeLany; Lidiya S Orlichenko; Catherine J Baty; Sruti S Shiva; Chandra Durgampudi; Jenny M Karlsson; Kenneth Lee; Kyongtae T Bae; Alessandro Furlan; Jaideep Behari; Shiguang Liu; Teresa McHale; Larry Nichols; Georgios Ioannis Papachristou; Dhiraj Yadav; Vijay P Singh
Journal:  Sci Transl Med       Date:  2011-11-02       Impact factor: 17.956

3.  Acute lipotoxicity regulates severity of biliary acute pancreatitis without affecting its initiation.

Authors:  Chandra Durgampudi; Pawan Noel; Krutika Patel; Rachel Cline; Ram N Trivedi; James P DeLany; Dhiraj Yadav; Georgios I Papachristou; Kenneth Lee; Chathur Acharya; Deepthi Jaligama; Sarah Navina; Faris Murad; Vijay P Singh
Journal:  Am J Pathol       Date:  2014-06       Impact factor: 4.307

Review 4.  Issues in hypertriglyceridemic pancreatitis: an update.

Authors:  John Scherer; Vijay P Singh; C S Pitchumoni; Dhiraj Yadav
Journal:  J Clin Gastroenterol       Date:  2014-03       Impact factor: 3.062

5.  Transcriptional regulation of CXC-ELR chemokines KC and MIP-2 in mouse pancreatic acini.

Authors:  Lidiya S Orlichenko; Jaideep Behari; Tzu-Hsuan Yeh; Shiguang Liu; Donna B Stolz; Ashok K Saluja; Vijay P Singh
Journal:  Am J Physiol Gastrointest Liver Physiol       Date:  2010-07-29       Impact factor: 4.052

6.  Fatty Acid Ethyl Esters Are Less Toxic Than Their Parent Fatty Acids Generated during Acute Pancreatitis.

Authors:  Krutika Patel; Chandra Durgampudi; Pawan Noel; Ram N Trivedi; Cristiane de Oliveira; Vijay P Singh
Journal:  Am J Pathol       Date:  2016-02-12       Impact factor: 4.307

7.  Peripancreatic fat necrosis worsens acute pancreatitis independent of pancreatic necrosis via unsaturated fatty acids increased in human pancreatic necrosis collections.

Authors:  Pawan Noel; Krutika Patel; Chandra Durgampudi; Ram N Trivedi; Cristiane de Oliveira; Michael D Crowell; Rahul Pannala; Kenneth Lee; Randall Brand; Jennifer Chennat; Adam Slivka; Georgios I Papachristou; Asif Khalid; David C Whitcomb; James P DeLany; Rachel A Cline; Chathur Acharya; Deepthi Jaligama; Faris M Murad; Dhiraj Yadav; Sarah Navina; Vijay P Singh
Journal:  Gut       Date:  2014-12-10       Impact factor: 23.059

8.  Relationship between carbachol hyperstimulation-induced pancreatic intracellular trypsinogen and NF-kappa B activation in rats in vitro.

Authors:  Chunfang Jiang; Hai Zheng; Sunan Liu; Kaifeng Fang
Journal:  J Huazhong Univ Sci Technolog Med Sci       Date:  2008-02

9.  Effects of increased intracellular cAMP on carbachol-stimulated zymogen activation, secretion, and injury in the pancreatic acinar cell.

Authors:  A Chaudhuri; T R Kolodecik; F S Gorelick
Journal:  Am J Physiol Gastrointest Liver Physiol       Date:  2004-09-30       Impact factor: 4.052

Review 10.  Pancreatitis and calcium signalling: report of an international workshop.

Authors:  Robert Sutton; Ole H Petersen; Stephen J Pandol
Journal:  Pancreas       Date:  2008-05       Impact factor: 3.327

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