Literature DB >> 11373422

Endothelial-derived selectins in the development of organ dysfunction in acute pancreatitis.

J J Powell1, A K Siriwardena, K C Fearon, J A Ross.   

Abstract

OBJECTIVE: The development of organ dysfunction is the principal determinant of outcome in acute pancreatitis and is mediated through a systemic inflammatory response characterized by leukocyte and endothelial cell activation. Up-regulation of the endothelial cell adhesion molecules, E-selectin and P-selectin, is important for endothelial/leukocyte interactions. Levels of serum-soluble E-selectin and P-selectin have been suggested as markers of endothelial activation. This study examines the kinetics of serum-soluble selectins in patients with acute pancreatitis complicated by organ dysfunction.
DESIGN: Prospective observational study.
SETTING: University teaching hospital with a specialist hepato-pancreatico-biliary service. PATIENTS: Eighteen patients with acute pancreatitis were studied, nine of whom had organ dysfunction.
MEASUREMENTS AND MAIN RESULTS: Serial venous blood samples were collected on the first 3 days after admission for measurement of soluble E-selectin and P-selectin by enzyme-linked immunosorbent assay. In all patients, soluble P-selectin concentrations decreased significantly during the study period. Nonsurvivors had significantly higher levels of soluble P-selectin than survivors. In contrast, soluble E-selectin increased significantly during the study period in patients with organ dysfunction, whereas it remained constant in patients without evidence of organ dysfunction.
CONCLUSIONS: These results suggest a role for endothelial-derived selectins in the development of organ dysfunction in patients with acute pancreatitis. The observed temporal differences in serum selectin concentrations is in keeping with in vitro observations of endothelial selectin expression.

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Year:  2001        PMID: 11373422     DOI: 10.1097/00003246-200103000-00018

Source DB:  PubMed          Journal:  Crit Care Med        ISSN: 0090-3493            Impact factor:   7.598


  7 in total

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2.  Microcirculation disturbance affects rats with acute severe pancreatitis following lung injury.

Authors:  Xue-Min Liu; Qing-Guang Liu; Jun Xu; Cheng-En Pan
Journal:  World J Gastroenterol       Date:  2005-10-21       Impact factor: 5.742

3.  The profiles of soluble adhesion molecules in the "great obstetrical syndromes".

Authors:  Nikolina Docheva; Roberto Romero; Piya Chaemsaithong; Adi L Tarca; Gaurav Bhatti; Percy Pacora; Bogdan Panaitescu; Noppadol Chaiyasit; Tinnakorn Chaiworapongsa; Eli Maymon; Sonia S Hassan; Offer Erez
Journal:  J Matern Fetal Neonatal Med       Date:  2018-02-01

Review 4.  Pathophysiological mechanisms in acute pancreatitis: Current understanding.

Authors:  Pankaj Singh; Pramod Kumar Garg
Journal:  Indian J Gastroenterol       Date:  2016-05-21

5.  Therapeutic efficacy of high-dose vitamin C on acute pancreatitis and its potential mechanisms.

Authors:  Wei-Dong Du; Zu-Rong Yuan; Jian Sun; Jian-Xiong Tang; Ai-Qun Cheng; Da-Ming Shen; Chun-Jin Huang; Xiao-Hua Song; Xiao-Feng Yu; Song-Bai Zheng
Journal:  World J Gastroenterol       Date:  2003-11       Impact factor: 5.742

6.  Activated Protein C Does Not Alleviate the Course of Systemic Inflammation in the APCAP Trial.

Authors:  Lea Kyhälä; Panu Mentula; Leena Kylänpää; Eeva Moilanen; Pauli Puolakkainen; Ville Pettilä; Heikki Repo
Journal:  Int J Inflam       Date:  2012-05-08

Review 7.  The Interplay between Inflammation, Coagulation and Endothelial Injury in the Early Phase of Acute Pancreatitis: Clinical Implications.

Authors:  Paulina Dumnicka; Dawid Maduzia; Piotr Ceranowicz; Rafał Olszanecki; Ryszard Drożdż; Beata Kuśnierz-Cabala
Journal:  Int J Mol Sci       Date:  2017-02-08       Impact factor: 5.923

  7 in total

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