Literature DB >> 11337620

Pineal transplantation after pinealectomy in young chickens has no effect on the development of scoliosis.

K M Bagnall1, M Beuerlein, P Johnson, J Wilson, V J Raso, M Moreau.   

Abstract

STUDY
DESIGN: Three experimental groups and one control group of chickens underwent different surgical procedures to determine the effects of pineal gland transplantation on the development of scoliosis.
OBJECTIVE: To determine whether transplantation of the pineal gland to the body wall musculature maintains serum melatonin levels at normal values and prevents the development of scoliosis. SUMMARY OF BACKGROUND DATA: Scoliosis occurs consistently after pinealectomy in young chickens. Many characteristics of this scoliosis are similar to those seen in patients with adolescent idiopathic scoliosis. It is not clear whether the underlying mechanism is dependent on reduced levels of serum melatonin or some other aspect of the extensive surgery.
METHODS: Four groups of chickens were selected: normal chickens, pinealectomized chickens, chickens that underwent simple cutting of the pineal stalk, and chickens that underwent transplantation of the pineal gland into the body wall. Development of scoliosis was determined from measurement of the Cobb angle from weekly radiographs.
RESULTS: All of the experimental groups showed the same levels of incidence and the same patterns of scoliosis development. Serum melatonin levels were reduced to nearly zero in all the experimental groups for the duration of the experiment. Scoliosis developed in none of the normal chickens.
CONCLUSIONS: Neither transplantation of the pineal gland into the body wall musculature nor simple cutting of the pineal stalk was able to maintain normal levels of serum melatonin because both procedures reduced levels to nearly zero. The incidence and pattern of scoliosis development in these groups were the same as those for the pinealectomized group. Reduction of serum melatonin levels remains a prerequisite for scoliosis development in young chickens.

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Year:  2001        PMID: 11337620     DOI: 10.1097/00007632-200105010-00007

Source DB:  PubMed          Journal:  Spine (Phila Pa 1976)        ISSN: 0362-2436            Impact factor:   3.468


  10 in total

1.  Evaluation of GPR50, hMel-1B, and ROR-alpha melatonin-related receptors and the etiology of adolescent idiopathic scoliosis.

Authors:  William Shyy; Kai Wang; Christina A Gurnett; Matthew B Dobbs; Nancy H Miller; Carol Wise; Val C Sheffield; Jose A Morcuende
Journal:  J Pediatr Orthop       Date:  2010-09       Impact factor: 2.324

Review 2.  [Molecular and genetic aspects of idiopathic scoliosis. Blood test for idiopathic scoliosis].

Authors:  A Moreau; M-Y Akoumé Ndong; B Azeddine; A Franco; P H Rompré; M-H Roy-Gagnon; I Turgeon; D Wang; K M Bagnall; B Poitras; H Labelle; C-H Rivard; G Grimard; J Ouellet; S Parent; F Moldovan
Journal:  Orthopade       Date:  2009-02       Impact factor: 1.087

3.  Understanding genetic factors in idiopathic scoliosis, a complex disease of childhood.

Authors:  Carol A Wise; Xiaochong Gao; Scott Shoemaker; Derek Gordon; John A Herring
Journal:  Curr Genomics       Date:  2008-03       Impact factor: 2.236

4.  Pinealectomy in the chicken: a good model of scoliosis?

Authors:  Andrew B Fagan; David J Kennaway; Andrew P Oakley
Journal:  Eur Spine J       Date:  2009-04-02       Impact factor: 3.134

5.  The effect of calmodulin antagonists on scoliosis: bipedal C57BL/6 mice model.

Authors:  Ibrahim Akel; Gokhan Demirkiran; Ahmet Alanay; Sevilay Karahan; Ralph Marcucio; Emre Acaroglu
Journal:  Eur Spine J       Date:  2009-02-26       Impact factor: 3.134

6.  Selective estrogen receptor modulation prevents scoliotic curve progression: radiologic and histomorphometric study on a bipedal C57Bl6 mice model.

Authors:  Gokhan Demirkiran; Ozgur Dede; Nadir Yalcin; Ibrahim Akel; Ralph Marcucio; Emre Acaroglu
Journal:  Eur Spine J       Date:  2013-10-18       Impact factor: 3.134

7.  Scientific basis for the potential use of melatonin in bone diseases: osteoporosis and adolescent idiopathic scoliosis.

Authors:  E J Sánchez-Barceló; M D Mediavilla; D X Tan; R J Reiter
Journal:  J Osteoporos       Date:  2010-06-01

Review 8.  The metabolic basis of adolescent idiopathic scoliosis: 2011 report of the "metabolic" workgroup of the Fondation Yves Cotrel.

Authors:  Emre Acaroglu; Regis Bobe; Jocelyn Enouf; Ralph Marcucio; Florina Moldovan; Alain Moreau
Journal:  Eur Spine J       Date:  2012-03-09       Impact factor: 3.134

Review 9.  A review of pinealectomy-induced melatonin-deficient animal models for the study of etiopathogenesis of adolescent idiopathic scoliosis.

Authors:  Gene Chi Wai Man; Man Gene Chi Wai; William Wei Jun Wang; Wang William Wei Jun; Annie Po Yee Yim; Yim Annie Po Yee; Jack Ho Wong; Wong Jack Ho; Tzi Bun Ng; Ng Tzi Bun; Tsz Ping Lam; Lam Tsz Ping; Simon Kwong Man Lee; Lee Simon Kwong Man; Bobby Kin Wah Ng; Ng Bobby Kin Wah; Chi Chiu Wang; Wang Chi Chiu; Yong Qiu; Qiu Yong; Chun Yiu Cheng; Cheng Jack Chun Yiu
Journal:  Int J Mol Sci       Date:  2014-09-18       Impact factor: 5.923

10.  Bioelectric activity in the suprachiasmatic nucleus-pineal gland system in children with adolescent idiopathic scoliosis.

Authors:  Dmitry Yu Pinchuk; Sergey S Bekshaev; Svetlana A Bumakova; Mikhail G Dudin; Olga D Pinchuk
Journal:  ISRN Orthop       Date:  2012-08-28
  10 in total

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