Literature DB >> 11293661

Hypothesis: is a failure to prevent bacteriolysis and the synergy among microbial and host-derived pro-inflammatory agonists the main contributory factors to the pathogenesis of post-infectious sequelae?

I Ginsburg1.   

Abstract

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Year:  2001        PMID: 11293661     DOI: 10.1023/a:1007024809661

Source DB:  PubMed          Journal:  Inflammation        ISSN: 0360-3997            Impact factor:   4.092


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  43 in total

Review 1.  Granulocyte colony-stimulating factor to prevent the progression of systemic nonresponsiveness in systemic inflammatory response syndrome and sepsis.

Authors:  M Weiss; L L Moldawer; E M Schneider
Journal:  Blood       Date:  1999-01-15       Impact factor: 22.113

Review 2.  Treating patients with severe sepsis.

Authors:  A P Wheeler; G R Bernard
Journal:  N Engl J Med       Date:  1999-01-21       Impact factor: 91.245

3.  Interleukin-10: the ambiguity in sepsis continues.

Authors:  P E Parsons
Journal:  Crit Care Med       Date:  1998-05       Impact factor: 7.598

Review 4.  Clinical trials in sepsis: where do we stand?

Authors:  J L Vincent
Journal:  J Crit Care       Date:  1997-03       Impact factor: 3.425

Review 5.  Antiendotoxin strategies for the prevention and treatment of septic shock. New approaches and future directions.

Authors:  S M Opal; R L Yu
Journal:  Drugs       Date:  1998-04       Impact factor: 9.546

Review 6.  The significance of endotoxin release in experimental and clinical sepsis in surgical patients--evidence for antibiotic-induced endotoxin release?

Authors:  R G Holzheimer
Journal:  Infection       Date:  1998 Mar-Apr       Impact factor: 3.553

7.  Tyrphostin AG 556 improves survival and reduces multiorgan failure in canine Escherichia coli peritonitis.

Authors:  J E Sevransky; G Shaked; A Novogrodsky; A Levitzki; A Gazit; A Hoffman; R J Elin; Z M Quezado; B D Freeman; P Q Eichacker; R L Danner; S M Banks; J Bacher; M L Thomas; C Natanson
Journal:  J Clin Invest       Date:  1997-04-15       Impact factor: 14.808

Review 8.  Cell damage in inflammatory and infectious sites might involve a coordinated "cross-talk" among oxidants, microbial haemolysins and ampiphiles, cationic proteins, phospholipases, fatty acids, proteinases and cytokines (an overview).

Authors:  I Ginsburg; R Kohen
Journal:  Free Radic Res       Date:  1995-06

Review 9.  Mechanisms of cell and tissue injury induced by group A streptococci: relation to poststreptococcal sequelae.

Authors:  I Ginsburg
Journal:  J Infect Dis       Date:  1972-09       Impact factor: 5.226

10.  Inhibition of cell wall turnover and autolysis by vancomycin in a highly vancomycin-resistant mutant of Staphylococcus aureus.

Authors:  K Sieradzki; A Tomasz
Journal:  J Bacteriol       Date:  1997-04       Impact factor: 3.490

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  1 in total

1.  Cationic polyelectrolytes from leukocytes might kill bacteria by activating their autolytic systems: enigmatically, the relevance of this phenomenon to post-infectious sequelae is disregarded.

Authors:  Isaac Ginsburg
Journal:  Intensive Care Med       Date:  2002-08       Impact factor: 17.440

  1 in total

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