Literature DB >> 11141862

The sac-4 gene of Neisseria gonorrhoeae and co-existing chlamydial infection.

D J Phillips1, C Patrizio, A Moyes, H Young.   

Abstract

BACKGROUND/
OBJECTIVES: Recently, the sac-4 gene in Neisseria gonorrhoeae was postulated to increase the risk of developing mixed gonococcal and chlamydial infection. The aims of this study were to determine the frequency of the sac-4 gene in a larger sample of isolates of different serovars and to assess the prevalence of sac-4 in gonococcal isolates from patients with and without coexisting chlamydial infection.
METHODS: Isolates from 259 episodes of gonorrhoea were tested by a PCR assay for the sac-4 gene. The presence of co-existing chlamydial infection was determined from both laboratory and GUM clinical records.
RESULTS: The overall prevalence of sac-4 was 57.5% (149/259). The prevalence was not the same in all serovars and ranged from 34.9% in serovar 1B2 to 100% in serovar 1B18. Exact logistic regression analysis indicated significant differences in sac-4 prevalence in isolates of different serovars. The prevalence of sac-4 was 69.5% (41/59) in gonococcal isolates from patients with co-existing chlamydial infection compared with 57.9% (62/107) for those without chlamydial infection. Exact logistic regression analysis showed that the slightly increased sac-4 prevalence among chlamydia positive patients (p = 0.2) virtually disappeared when serovar status was taken into account (p > 0.9).
CONCLUSION: The sac-4 gene of the gonococcus does not increase the risk for mixed chlamydial infection.

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Year:  2000        PMID: 11141862      PMCID: PMC1744224          DOI: 10.1136/sti.76.5.400

Source DB:  PubMed          Journal:  Sex Transm Infect        ISSN: 1368-4973            Impact factor:   3.519


  3 in total

1.  Epidemiology and transmission patterns of concomitant genital chlamydial and gonococcal infections.

Authors:  P Matondo; I Johnson; S Sivapalan
Journal:  Genitourin Med       Date:  1995-08

2.  Pelvic inflammatory disease isolates of Neisseria gonorrhoeae are distinguished by C1q-dependent virulence for newborn rats and by the sac-4 region.

Authors:  S Nowicki; P Ram; T Pham; P Goluszko; S Morse; G D Anderson; B Nowicki
Journal:  Infect Immun       Date:  1997-06       Impact factor: 3.441

3.  Gonococcal infection in a nonhuman host is determined by human complement C1q.

Authors:  S Nowicki; M G Martens; B J Nowicki
Journal:  Infect Immun       Date:  1995-12       Impact factor: 3.441

  3 in total
  1 in total

1.  Trends and determinants of antimicrobial susceptibility of Neisseria gonorrhoeae in the Netherlands, 2007 to 2015.

Authors:  Sanne Hi Hofstraat; Hannelore M Götz; Alje P van Dam; Marianne Ab van der Sande; Birgit Hb van Benthem
Journal:  Euro Surveill       Date:  2018-09
  1 in total

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