Literature DB >> 11135610

NO vascular control in Duchenne muscular dystrophy.

R H Crosbie1.   

Abstract

A new investigation into Duchenne muscular dystrophy (DMD) pathogenesis suggests that at least part of the muscle degeneration observed in DMD patients may result from the reduced production of muscle membrane-associated neuronal nitric oxide synthase. This reduction may lead to impaired regulation of the vasoconstrictor response and eventual muscle damage.

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Year:  2001        PMID: 11135610     DOI: 10.1038/83309

Source DB:  PubMed          Journal:  Nat Med        ISSN: 1078-8956            Impact factor:   53.440


  4 in total

1.  Flt-1 haploinsufficiency ameliorates muscular dystrophy phenotype by developmentally increased vasculature in mdx mice.

Authors:  Mayank Verma; Yoko Asakura; Hiroyuki Hirai; Shuichi Watanabe; Christopher Tastad; Guo-Hua Fong; Masatsugu Ema; Jarrod A Call; Dawn A Lowe; Atsushi Asakura
Journal:  Hum Mol Genet       Date:  2010-08-12       Impact factor: 6.150

Review 2.  Therapeutic aspects of cell signaling and communication in Duchenne muscular dystrophy.

Authors:  Alicja Starosta; Patryk Konieczny
Journal:  Cell Mol Life Sci       Date:  2021-04-07       Impact factor: 9.261

Review 3.  Muscle fatigue, nNOS and muscle fiber atrophy in limb girdle muscular dystrophy.

Authors:  Corrado Angelini; Elisabetta Tasca; Anna Chiara Nascimbeni; Marina Fanin
Journal:  Acta Myol       Date:  2014-12

Review 4.  Fatigue in muscular dystrophies.

Authors:  Corrado Angelini; Elisabetta Tasca
Journal:  Neuromuscul Disord       Date:  2012-12       Impact factor: 4.296

  4 in total

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