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Literature DB >> 11122749

Postprandial lipemia and coronary risk.

W Patsch¹, H Esterbauer, B Föger, J R Patsch.

Abstract

A number of cross-sectional studies have demonstrated that the magnitude of postprandial lipemia or single postprandial triglyceride values predict asymptomatic and symptomatic atherosclerosis, independent of risk factors measured in the fasting state. Postprandial lipemia reflects an integrated measure of an individual's triglyceride metabolic capacity. Numerous genetic and environmental factors that are known or suspected to affect triglyceride transport contribute to the magnitude of postprandial lipemia. In this article, mechanisms linking postprandial lipemia with the development and progression of atherosclerosis are described, and determinants of the extent and duration of postprandial lipemia are discussed.

Entities: Chemical Disease

Mesh：

Substances：
Triglycerides

Year: 2000 PMID： 11122749 DOI： 10.1007/s11883-000-0025-0

Source DB: PubMed Journal: Curr Atheroscler Rep ISSN： 1523-3804 Impact factor: 5.113

151 in total

1. Lipoprotein lipase gene variation is associated with a paternal history of premature coronary artery disease and fasting and postprandial plasma triglycerides: the European Atherosclerosis Research Study (EARS).

Authors: S E Humphries; V Nicaud; J Margalef; L Tiret; P J Talmud
Journal: Arterioscler Thromb Vasc Biol Date: 1998-04 Impact factor: 8.311

2. Possible functional interactions of apolipoprotein B-100 segments that associate with cell proteoglycans and the ApoB/E receptor.

Authors: U Olsson; G Camejo; E Hurt-Camejo; K Elfsber; O Wiklund; G Bondjers
Journal: Arterioscler Thromb Vasc Biol Date: 1997-01 Impact factor: 8.311

3. Postprandial plasma glucose, insulin, glucagon and triglyceride responses to a standard diet in normal subjects.

Authors: M Ahmed; M C Gannon; F Q Nuttall
Journal: Diabetologia Date: 1976-03 Impact factor: 10.122

4. Purification and characterization of acylation stimulating protein.

Authors: K M Cianflone; A D Sniderman; M J Walsh; H T Vu; J Gagnon; M A Rodriguez
Journal: J Biol Chem Date: 1989-01-05 Impact factor: 5.157

5. Common genetic variation in the promoter of the human apo CIII gene abolishes regulation by insulin and may contribute to hypertriglyceridemia.

Authors: W W Li; M M Dammerman; J D Smith; S Metzger; J L Breslow; T Leff
Journal: J Clin Invest Date: 1995-12 Impact factor: 14.808

6. Deficiency of cholesteryl ester transfer protein. Description of the molecular defect and the dissociation of cholesteryl ester and triglyceride transport in plasma.

Authors: A Ritsch; H Drexel; F W Amann; C Pfeifhofer; J R Patsch
Journal: Arterioscler Thromb Vasc Biol Date: 1997-12 Impact factor: 8.311

7. Hypertriglyceridemia as a result of human apo CIII gene expression in transgenic mice.

Authors: Y Ito; N Azrolan; A O'Connell; A Walsh; J L Breslow
Journal: Science Date: 1990-08-17 Impact factor: 47.728

8. Effect of apo E phenotype on plasma postprandial triglyceride levels in young male adults with and without a familial history of myocardial infarction: the EARS II study. European Atherosclerosis Research Study.

Authors: J Dallongeville; L Tiret; S Visvikis; D S O'Reilly; M Saava; G Tsitouris; M Rosseneu; G DeBacker; S E Humphries; U Beisiegel
Journal: Atherosclerosis Date: 1999-08 Impact factor: 5.162

Postprandial lipemia and coronary risk.

1. Lipoprotein lipase gene variation is associated with a paternal history of premature coronary artery disease and fasting and postprandial plasma triglycerides: the European Atherosclerosis Research Study (EARS).

2. Possible functional interactions of apolipoprotein B-100 segments that associate with cell proteoglycans and the ApoB/E receptor.

3. Postprandial plasma glucose, insulin, glucagon and triglyceride responses to a standard diet in normal subjects.

4. Purification and characterization of acylation stimulating protein.

5. Common genetic variation in the promoter of the human apo CIII gene abolishes regulation by insulin and may contribute to hypertriglyceridemia.

6. Deficiency of cholesteryl ester transfer protein. Description of the molecular defect and the dissociation of cholesteryl ester and triglyceride transport in plasma.

7. Hypertriglyceridemia as a result of human apo CIII gene expression in transgenic mice.

8. Effect of apo E phenotype on plasma postprandial triglyceride levels in young male adults with and without a familial history of myocardial infarction: the EARS II study. European Atherosclerosis Research Study.

9. Enhanced triglyceride clearance with intraperitoneal human acylation stimulating protein in C57BL/6 mice.

10. Mechanism of plasma cholesteryl ester transfer in hypertriglyceridemia.

Review 1. Triglyceride Treatment in the Age of Cholesterol Reduction.

2. The influence of walking performed immediately before meals with moderate fat content on postprandial lipemia.

3. Epigenome-wide association study of triglyceride postprandial responses to a high-fat dietary challenge.

Review 4. Role of Fluid Milk in Attenuating Postprandial Hyperglycemia and Hypertriglyceridemia.