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Literature DB >> 11110766

Ventricular fibrillation: how do we stop the waves from breaking?

J N Weiss¹, P S Chen, Z Qu, H S Karagueuzian, A Garfinkel.

Abstract

Combined experimental and theoretical developments have demonstrated that in addition to preexisting electrophysiological heterogeneities, cardiac electrical restitution properties contribute to breakup of reentrant wavefronts during cardiac fibrillation. Developing therapies that favorably alter electrical restitution properties have promise as a new paradigm for preventing fibrillation.

Entities: Disease

Mesh：

Substances：
Anti-Arrhythmia Agents

Year: 2000 PMID： 11110766 DOI： 10.1161/01.res.87.12.1103

Source DB: PubMed Journal: Circ Res ISSN： 0009-7330 Impact factor: 17.367

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Cited

42 in total

1. Panoramic optical mapping shows wavebreak at a consistent anatomical site at the onset of ventricular fibrillation.

Authors: Elliot B Bourgeois; Hugh D Reeves; Gregory P Walcott; Jack M Rogers
Journal: Cardiovasc Res Date: 2011-12-05 Impact factor: 10.787

2. Critical mass hypothesis revisited: role of dynamical wave stability in spontaneous termination of cardiac fibrillation.

Authors: Zhilin Qu
Journal: Am J Physiol Heart Circ Physiol Date: 2005-08-19 Impact factor: 4.733

3. Complex-periodic spiral waves in confluent cardiac cell cultures induced by localized inhomogeneities.

Authors: Seong-Min Hwang; Tae Yun Kim; Kyoung J Lee
Journal: Proc Natl Acad Sci U S A Date: 2005-06-28 Impact factor: 11.205

4. Conditions for propagation and block of excitation in an asymptotic model of atrial tissue.

Authors: Radostin D Simitev; Vadim N Biktashev
Journal: Biophys J Date: 2006-01-13 Impact factor: 4.033

5. A case of short-coupled variant of torsade de pointes characterized by spatial heterogeneity of action potential duration and its restitution kinetics.

Authors: Masatoshi Yamazaki; Toshiyuki Osaka; Eriko Yokoyama; Itsuo Kodama
Journal: J Interv Card Electrophysiol Date: 2007-01-26 Impact factor: 1.900

10. Criteria for arrhythmogenicity in genetically-modified Langendorff-perfused murine hearts modelling the congenital long QT syndrome type 3 and the Brugada syndrome.

Authors: Ian N Sabir; Lucia M Li; Victoria J Jones; Catharine A Goddard; Andrew A Grace; Christopher L-H Huang
Journal: Pflugers Arch Date: 2007-09-06 Impact factor: 3.657

Ventricular fibrillation: how do we stop the waves from breaking?

1. Panoramic optical mapping shows wavebreak at a consistent anatomical site at the onset of ventricular fibrillation.

2. Critical mass hypothesis revisited: role of dynamical wave stability in spontaneous termination of cardiac fibrillation.

3. Complex-periodic spiral waves in confluent cardiac cell cultures induced by localized inhomogeneities.

4. Conditions for propagation and block of excitation in an asymptotic model of atrial tissue.

5. A case of short-coupled variant of torsade de pointes characterized by spatial heterogeneity of action potential duration and its restitution kinetics.

6. How does β-adrenergic signalling affect the transitions from ventricular tachycardia to ventricular fibrillation?

7. Acute amiodarone promotes drift and early termination of spiral wave re-entry.

8. Selective inhibition of late sodium current suppresses ventricular tachycardia and fibrillation in intact rat hearts.

Review 9. Ventricular fibrillation and defibrillation.

10. Criteria for arrhythmogenicity in genetically-modified Langendorff-perfused murine hearts modelling the congenital long QT syndrome type 3 and the Brugada syndrome.