Literature DB >> 11094073

Vav3 mediates receptor protein tyrosine kinase signaling, regulates GTPase activity, modulates cell morphology, and induces cell transformation.

L Zeng1, P Sachdev, L Yan, J L Chan, T Trenkle, M McClelland, J Welsh, L H Wang.   

Abstract

A recently reported new member of the Vav family proteins, Vav3 has been identified as a Ros receptor protein tyrosine kinase (RPTK) interacting protein by yeast two-hybrid screening. Northern analysis shows that Vav3 has a broad tissue expression profile that is distinct from those of Vav and Vav2. Two species of Vav3 transcripts, 3.4 and 5.4 kb, were detected with a differential expression pattern in various tissues. Transient expression of Vav in 293T and NIH 3T3 cells demonstrated that ligand stimulation of several RPTKs (epidermal growth factor receptor [EGFR], Ros, insulin receptor [IR], and insulin-like growth factor I receptor [IGFR]) led to tyrosine phosphorylation of Vav3 and its association with the receptors as well as their downstream signaling molecules, including Shc, Grb2, phospholipase C (PLC-gamma), and phosphatidylinositol 3 kinase. In vitro binding assays using glutathione S-transferase-fusion polypeptides containing the GTPase-binding domains of Rok-alpha, Pak, or Ack revealed that overexpression of Vav3 in NIH 3T3 cells resulted in the activation of Rac-1 and Cdc42 whereas a deletion mutant lacking the N-terminal calponin homology and acidic region domains activated RhoA and Rac-1 but lost the ability to activate Cdc42. Vav3 induced marked membrane ruffles and microspikes in NIH 3T3 cells, while the N-terminal truncation mutants of Vav3 significantly enhanced membrane ruffle formation but had a reduced ability to induce microspikes. Activation of IR further enhanced the ability of Vav3 to induce membrane ruffles, but IGFR activation specifically promoted Vav3-mediated microspike formation. N-terminal truncation of Vav3 activated its transforming potential, as measured by focus-formation assays. We conclude that Vav3 mediates RPTK signaling and regulates GTPase activity, its native and mutant forms are able to modulate cell morphology, and it has the potential to induce cell transformation.

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Year:  2000        PMID: 11094073      PMCID: PMC102179          DOI: 10.1128/MCB.20.24.9212-9224.2000

Source DB:  PubMed          Journal:  Mol Cell Biol        ISSN: 0270-7306            Impact factor:   4.272


  63 in total

1.  CD19 as a membrane-anchored adaptor protein of B lymphocytes: costimulation of lipid and protein kinases by recruitment of Vav.

Authors:  L M O'Rourke; R Tooze; M Turner; D M Sandoval; R H Carter; V L Tybulewicz; D T Fearon
Journal:  Immunity       Date:  1998-05       Impact factor: 31.745

2.  Signal transduction in T lymphocytes using a conditional allele of Sos.

Authors:  L J Holsinger; D M Spencer; D J Austin; S L Schreiber; G R Crabtree
Journal:  Proc Natl Acad Sci U S A       Date:  1995-10-10       Impact factor: 11.205

3.  The proline-rich region of Vav binds to Grb2 and Grb3-3.

Authors:  F Ramos-Morales; F Romero; F Schweighoffer; G Bismuth; J Camonis; M Tortolero; S Fischer
Journal:  Oncogene       Date:  1995-10-19       Impact factor: 9.867

4.  Vav binds to several SH2/SH3 containing proteins in activated lymphocytes.

Authors:  F Ramos-Morales; B J Druker; S Fischer
Journal:  Oncogene       Date:  1994-07       Impact factor: 9.867

5.  Hematopoietic development of vav-/- mouse embryonic stem cells.

Authors:  R Zhang; F Y Tsai; S H Orkin
Journal:  Proc Natl Acad Sci U S A       Date:  1994-12-20       Impact factor: 11.205

6.  Binding of Vav to Grb2 through dimerization of Src homology 3 domains.

Authors:  Z S Ye; D Baltimore
Journal:  Proc Natl Acad Sci U S A       Date:  1994-12-20       Impact factor: 11.205

7.  Identification of VAV2 on 9q34 and its exclusion as the tuberous sclerosis gene TSC1.

Authors:  E P Henske; M P Short; S Jozwiak; C M Bovey; S Ramlakhan; J L Haines; D J Kwiatkowski
Journal:  Ann Hum Genet       Date:  1995-01       Impact factor: 1.670

8.  Insulin-like growth factor-1 induces rapid tyrosine phosphorylation of the vav proto-oncogene product.

Authors:  S Uddin; A Yetter; S Katzav; C Hofmann; M F White; L C Platanias
Journal:  Exp Hematol       Date:  1996-04       Impact factor: 3.084

9.  Insulin-dependent tyrosine phosphorylation of the vav protooncogene product in cells of hematopoietic origin.

Authors:  S Uddin; S Katzav; M F White; L C Platanias
Journal:  J Biol Chem       Date:  1995-03-31       Impact factor: 5.157

10.  A functional T-cell receptor signaling pathway is required for p95vav activity.

Authors:  J Wu; S Katzav; A Weiss
Journal:  Mol Cell Biol       Date:  1995-08       Impact factor: 4.272

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  51 in total

1.  Vav3 is regulated during the cell cycle and effects cell division.

Authors:  Keiko Fujikawa; Yoshiro Inoue; Masaharu Sakai; Yoshikazu Koyama; Shinzo Nishi; Ryo Funada; Frederick W Alt; Wojciech Swat
Journal:  Proc Natl Acad Sci U S A       Date:  2002-03-26       Impact factor: 11.205

2.  Critical but distinct roles for the pleckstrin homology and cysteine-rich domains as positive modulators of Vav2 signaling and transformation.

Authors:  Michelle A Booden; Sharon L Campbell; Channing J Der
Journal:  Mol Cell Biol       Date:  2002-04       Impact factor: 4.272

3.  Essential role for vav Guanine nucleotide exchange factors in brain-derived neurotrophic factor-induced dendritic spine growth and synapse plasticity.

Authors:  Carly F Hale; Karen C Dietz; Juan A Varela; Cody B Wood; Benjamin C Zirlin; Leah S Leverich; Robert W Greene; Christopher W Cowan
Journal:  J Neurosci       Date:  2011-08-31       Impact factor: 6.167

4.  RACK1, an insulin-like growth factor I (IGF-I) receptor-interacting protein, modulates IGF-I-dependent integrin signaling and promotes cell spreading and contact with extracellular matrix.

Authors:  Ulrich Hermanto; Cong S Zong; Weiqun Li; Lu-Hai Wang
Journal:  Mol Cell Biol       Date:  2002-04       Impact factor: 4.272

Review 5.  Cell growth and metastasis in pancreatic cancer: is Vav the Rho'd to activation?

Authors:  Daniel D Billadeau
Journal:  Int J Gastrointest Cancer       Date:  2002

6.  Targeted overexpression of vav3 oncogene in prostatic epithelium induces nonbacterial prostatitis and prostate cancer.

Authors:  Yin Liu; Jun Qin Mo; Qiande Hu; Gregory Boivin; Linda Levin; Shan Lu; Dianer Yang; Zhongyun Dong; Shan Lu
Journal:  Cancer Res       Date:  2008-08-01       Impact factor: 12.701

7.  Endogenous RhoG is rapidly activated after epidermal growth factor stimulation through multiple guanine-nucleotide exchange factors.

Authors:  Thomas Samson; Christopher Welch; Elizabeth Monaghan-Benson; Klaus M Hahn; Keith Burridge
Journal:  Mol Biol Cell       Date:  2010-03-17       Impact factor: 4.138

8.  Bypass mechanisms of the androgen receptor pathway in therapy-resistant prostate cancer cell models.

Authors:  Rute B Marques; Natasja F Dits; Sigrun Erkens-Schulze; Wytske M van Weerden; Guido Jenster
Journal:  PLoS One       Date:  2010-10-19       Impact factor: 3.240

Review 9.  Molecular pathways: ROS1 fusion proteins in cancer.

Authors:  Kurtis D Davies; Robert C Doebele
Journal:  Clin Cancer Res       Date:  2013-05-29       Impact factor: 12.531

10.  Vav3-deficient mice exhibit a transient delay in cerebellar development.

Authors:  Celia Quevedo; Vincent Sauzeau; Mauricio Menacho-Márquez; Antonio Castro-Castro; Xosé R Bustelo
Journal:  Mol Biol Cell       Date:  2010-01-20       Impact factor: 4.138

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