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Literature DB >> 11078764

Infection of breast epithelial cells with Epstein-Barr virus via cell-to-cell contact.

P Speck¹, R Longnecker.

Abstract

Entities: Species

Mesh：

Year: 2000 PMID： 11078764 DOI： 10.1093/jnci/92.22.1849

Source DB: PubMed Journal: J Natl Cancer Inst ISSN： 0027-8874 Impact factor: 13.506

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23 in total

1. A phase 1/2 trial of arginine butyrate and ganciclovir in patients with Epstein-Barr virus-associated lymphoid malignancies.

Authors: Susan P Perrine; Olivier Hermine; Trudy Small; Felipe Suarez; Richard O'Reilly; Farid Boulad; Joyce Fingeroth; Melissa Askin; Arthur Levy; Steven J Mentzer; Massimo Di Nicola; Alessandro M Gianni; Christoph Klein; Steven Horwitz; Douglas V Faller
Journal: Blood Date: 2006-11-21 Impact factor: 22.113

2. Cell-surface expression of a mutated Epstein-Barr virus glycoprotein B allows fusion independent of other viral proteins.

Authors: Marisa P McShane; Richard Longnecker
Journal: Proc Natl Acad Sci U S A Date: 2004-12-06 Impact factor: 11.205

3. Cleavage of Epstein-Barr virus glycoprotein B is required for full function in cell-cell fusion with both epithelial and B cells.

Authors: Jessica Sorem; Richard Longnecker
Journal: J Gen Virol Date: 2009-03 Impact factor: 3.891

4. Exosomes derived from Epstein-Barr virus-infected cells are internalized via caveola-dependent endocytosis and promote phenotypic modulation in target cells.

Authors: Asuka Nanbo; Eri Kawanishi; Ryuji Yoshida; Hironori Yoshiyama
Journal: J Virol Date: 2013-07-17 Impact factor: 5.103

Infection of breast epithelial cells with Epstein-Barr virus via cell-to-cell contact.

1. A phase 1/2 trial of arginine butyrate and ganciclovir in patients with Epstein-Barr virus-associated lymphoid malignancies.

2. Cell-surface expression of a mutated Epstein-Barr virus glycoprotein B allows fusion independent of other viral proteins.

3. Cleavage of Epstein-Barr virus glycoprotein B is required for full function in cell-cell fusion with both epithelial and B cells.

4. Exosomes derived from Epstein-Barr virus-infected cells are internalized via caveola-dependent endocytosis and promote phenotypic modulation in target cells.

Review 5. Current and potential treatments for ubiquitous but neglected herpesvirus infections.

6. Dysregulation of HER2/HER3 signaling axis in Epstein-Barr virus-infected breast carcinoma cells.

7. Roles of cell signaling pathways in cell-to-cell contact-mediated Epstein-Barr virus transmission.

8. Epstein-Barr virus infection in ex vivo tonsil epithelial cell cultures of asymptomatic carriers.

9. De novo infection of B cells during murine gammaherpesvirus 68 latency.

10. Case report of concurrent primary malignancies of the breast and nasopharynx.