Literature DB >> 11055602

Predictors of infectious complications after burn injuries in children.

G L Rodgers1, J Mortensen, M C Fisher, A Lo, A Cresswell, S S Long.   

Abstract

BACKGROUND: Infections are the major life-threatening complication of burn injury and occur with the greatest frequency in children. Knowledge of their occurrence and management, however, is extrapolated from studies in adults. We performed a prospective study of infectious complications in burned children.
OBJECTIVE: To delineate epidemiology, risk factors and microbiology of infections in burned children where burn care and surgical interventions are optimal.
METHODS: Children hospitalized for burns were entered into prospective study. Characteristics of the burn injury were assessed, and active surveillance for infections was performed.
RESULTS: Seventy patients were entered [mean age, 42 months; mean total body surface area (TBSA), burn 15%]. Twenty-seven percent of patients developed 39 infections: 13 involved the burn wound (burn wound sepsis, 6; graft loss, 5; and cellulitis, 2); 13 were catheter-associated septicemia; 13 involved other sites (i.e. pneumonia, 4; urinary tract infection, 3; bacteremia, 2; endocarditis, 1; myocardial abscess, 1; toxin-mediated syndrome, 1; and otitis media, 1). Twenty-three infections were caused by a single organism, 9 infections by more than 1 organism and in 7 infections defined by CDC criteria no organism was recovered. Organisms causing infection were: Staphylococcus aureus, 19; Candida albicans, 4; Pseudomonas aeruginosa, 4; coagulase-negative Staphylococcus, 4; Enterococcus sp., 3; Escherichia coli, 1; Klebsiella oxytoca, 1; Serratia marcescens, 1; Streptococcus pneumoniae, 1; Streptococcus pyogenes, 1; Aspergillus fumigatus, 1; and Candida parapsilosis, 1. Burn mechanism (flame and inhalation), extent (TBSA >30%) and depth (full thickness) were risk factors for infection; young age and site of burn were not.
CONCLUSION: The most common infections occurring in burn children are burn wound infections and catheter-associated septicemia. Characteristics of burn injury predict risk of infection. Children with flame and inhalation injury, TBSA burned >30% and full thickness burns are at high risk of infectious complications.

Entities:  

Mesh:

Year:  2000        PMID: 11055602     DOI: 10.1097/00006454-200010000-00010

Source DB:  PubMed          Journal:  Pediatr Infect Dis J        ISSN: 0891-3668            Impact factor:   2.129


  17 in total

1.  Septic mice are susceptible to pulmonary aspergillosis.

Authors:  Claudia F Benjamim; Cory M Hogaboam; Nicholas W Lukacs; Steven L Kunkel
Journal:  Am J Pathol       Date:  2003-12       Impact factor: 4.307

2.  2007 Guideline for Isolation Precautions: Preventing Transmission of Infectious Agents in Health Care Settings.

Authors:  Jane D Siegel; Emily Rhinehart; Marguerite Jackson; Linda Chiarello
Journal:  Am J Infect Control       Date:  2007-12       Impact factor: 2.918

3.  Dendritic cell modification of neutrophil responses to infection after burn injury.

Authors:  Julia Bohannon; Weihua Cui; Edward Sherwood; Tracy Toliver-Kinsky
Journal:  J Immunol       Date:  2010-08-02       Impact factor: 5.422

4.  Negative wound pressure therapy is safe and useful in pediatric burn patients.

Authors:  Yanhan Ren; Philip Chang; Robert L Sheridan
Journal:  Int J Burns Trauma       Date:  2017-04-15

5.  Insulin increases resistance to burn wound infection-associated sepsis.

Authors:  Gerd G Gauglitz; Tracy E Toliver-Kinsky; Felicia N Williams; Juquan Song; Weihua Cui; David N Herndon; Marc G Jeschke
Journal:  Crit Care Med       Date:  2010-01       Impact factor: 7.598

6.  Virulence potential of Escherichia coli isolates from skin and soft tissue infections.

Authors:  Ziva Petkovsek; Kristina Elersic; Marija Gubina; Darja Zgur-Bertok; Marjanca Starcic Erjavec
Journal:  J Clin Microbiol       Date:  2009-04-08       Impact factor: 5.948

7.  Fms-like tyrosine kinase-3 ligand alters antigen-specific responses to infections after severe burn injury.

Authors:  Julia Bohannon; Geping Fang; Weihua Cui; Edward Sherwood; Tracy Toliver-Kinsky
Journal:  Shock       Date:  2009-10       Impact factor: 3.454

8.  Stimulation of hematopoiesis by the Fms-like tyrosine kinase 3 ligand restores bacterial induction of Th1 cytokines in thermally injured mice.

Authors:  Tracy E Toliver-Kinsky; Cheng Y Lin; David N Herndon; Edward R Sherwood
Journal:  Infect Immun       Date:  2003-06       Impact factor: 3.441

9.  Innate immune function predicts the development of nosocomial infection in critically injured children.

Authors:  Jennifer A Muszynski; Ryan Nofziger; Kristin Greathouse; Jyotsna Nateri; Lisa Hanson-Huber; Lisa Steele; Kathleen Nicol; Jonathan I Groner; Gail E Besner; Corey Raffel; Susan Geyer; Osama El-Assal; Mark W Hall
Journal:  Shock       Date:  2014-10       Impact factor: 3.454

10.  Endogenous Fms-like tyrosine kinase-3 ligand levels are not altered in mice after a severe burn and infection.

Authors:  Julia K Bohannon; Weihua Cui; Tracy Toliver-Kinsky
Journal:  BMC Immunol       Date:  2009-08-28       Impact factor: 3.615

View more

北京卡尤迪生物科技股份有限公司 © 2022-2023.