PURPOSE: The goal of this study was to follow over time the MR imaging features of cortical laminar necrosis. PATIENTS AND METHODS: Six patients with cortical laminar necrosis were included. There were two women and four men aged 54-84 years, with a mean age of 68 years. In four patients, cortical laminar necrosis was caused by ischemic stroke, one case occurred after a cardiac arrest and the last patient had a meningoencephalitis. The time delay from insult to the first MR study varied between one week and 3 months. RESULTS: The MRI showed hyperintense lesions in the cerebral cortex on T1W and T2W images. The high intensity signal was still observed a few months after the insult. Cortical laminar necrosis lesions did not demonstrate hemorrhage on CT and MRI studies. CONCLUSION: MRI allowed detection of cortical laminar necrosis and could differentiate it from hemorrhage.
PURPOSE: The goal of this study was to follow over time the MR imaging features of cortical laminar necrosis. PATIENTS AND METHODS: Six patients with cortical laminar necrosis were included. There were two women and four men aged 54-84 years, with a mean age of 68 years. In four patients, cortical laminar necrosis was caused by ischemic stroke, one case occurred after a cardiac arrest and the last patient had a meningoencephalitis. The time delay from insult to the first MR study varied between one week and 3 months. RESULTS: The MRI showed hyperintense lesions in the cerebral cortex on T1W and T2W images. The high intensity signal was still observed a few months after the insult. Cortical laminar necrosis lesions did not demonstrate hemorrhage on CT and MRI studies. CONCLUSION: MRI allowed detection of cortical laminar necrosis and could differentiate it from hemorrhage.
Authors: K O Lövblad; S G Wetzel; T Somon; K Wilhelm; A Mehdizade; A Kelekis; M El-Koussy; S El-Tatawy; M Bishof; G Schroth; S Perrig; F Lazeyras; R Sztajzel; F Terrier; D Rüfenacht; J Delavelle Journal: Neuroradiology Date: 2004-01-29 Impact factor: 2.804