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6 in total

1. RhoA biological activity is dependent on prenylation but independent of specific isoprenoid modification.

Authors: Patricia A Solski; Whitney Helms; Patricia J Keely; Lishan Su; Channing J Der
Journal: Cell Growth Differ Date: 2002-08

2. Multiple sequence elements facilitate Chp Rho GTPase subcellular location, membrane association, and transforming activity.

Authors: Emily J Chenette; Natalia Y Mitin; Channing J Der
Journal: Mol Biol Cell Date: 2006-04-26 Impact factor: 4.138

3. Transformation by a nucleotide-activated P2Y receptor is mediated by activation of Galphai, Galphaq and Rho-dependent signaling pathways.

Authors: Anurag Singh; José L Boyer; Channing J Der; Irene E Zohn
Journal: J Mol Signal Date: 2010-07-23

4. DLC-1 suppresses non-small cell lung cancer growth and invasion by RhoGAP-dependent and independent mechanisms.

Authors: Kevin D Healy; Louis Hodgson; Tai-Young Kim; Adam Shutes; Savitri Maddileti; Rudolph L Juliano; Klaus M Hahn; T Kendall Harden; Yung-Jue Bang; Channing J Der
Journal: Mol Carcinog Date: 2008-05 Impact factor: 4.784

5. The nuclear RhoA exchange factor Net1 interacts with proteins of the Dlg family, affects their localization, and influences their tumor suppressor activity.

Authors: Rafael García-Mata; Adi D Dubash; Lisa Sharek; Heather S Carr; Jeffrey A Frost; Keith Burridge
Journal: Mol Cell Biol Date: 2007-10-15 Impact factor: 4.272

6. Romidepsin inhibits Ras-dependent growth transformation of NIH 3T3 fibroblasts and RIE-1 epithelial cells independently of Ras signaling inhibition.

Authors: Ariella B Hanker; Kevin D Healy; Jean Nichols; Channing J Der
Journal: J Mol Signal Date: 2009-08-16

6 in total