Literature DB >> 11032593

Homoharringtonine and low-dose cytarabine in the management of late chronic-phase chronic myelogenous leukemia.

H M Kantarjian1, M Talpaz, T L Smith, J Cortes, F J Giles, M B Rios, S Mallard, J Gajewski, A Murgo, B Cheson, S O'Brien.   

Abstract

PURPOSE: : To evaluate the efficacy and toxicity profiles of a combination regimen of homoharringtonine (HHT) and low-dose cytarabine (ara-C) in patients with Philadelphia chromosome (Ph)-positive chronic myelogenous leukemia (CML) who had experienced treatment failure with interferon alfa (IFNalpha) therapy. PATIENTS AND METHODS: One hundred five patients were treated: 100 in chronic phase (15 with cytogenetic clonal evolution) and five in accelerated phase. Their median age was 52 years; all had been treated unsuccessfully with IFNalpha; 94% were in late chronic phase; 43% had been exposed to ara-C and 11% had been exposed to HHT. Patients received HHT 2.5 mg/m(2) by continuous infusion daily for 5 days and ara-C 15mg/m(2) daily in two subcutaneous injections for 5 days every 4 weeks. The outcome of the 100 patients in chronic phase was compared with a previous study group of 73 patients treated with HHT alone.
RESULTS: Overall, the complete hematologic response (CHR) rate in chronic phase was 72%; the cytogenetic response rate was 32% (major response, 15%; complete response, 5%). Toxicities were acceptable, mostly related to moderate diarrhea (3%), headaches (3%), cardiovascular events (3%),and myelosuppression-associated complications (3% to 14%). With a median follow-up period of 25 months, the estimated 4-year survival rate was 55%. Response rates were identical with HHT plus ara-C versus HHT alone, but the survival was significantly longer with the combination after accounting for differences in the study groups and by multivariate analysis.
CONCLUSION: The combination regimen of HHT and ara-C is effective and safe in patients with CML who have experienced treatment failure with IFNalpha and needs to be investigated together with IFNalpha as part of front-line CML therapy. The addition of ara-C did not improve the response rates but may have improved survival, perhaps through suppression of clones related to disease transformation.

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Year:  2000        PMID: 11032593     DOI: 10.1200/JCO.2000.18.20.3513

Source DB:  PubMed          Journal:  J Clin Oncol        ISSN: 0732-183X            Impact factor:   44.544


  17 in total

1.  Homoharringtonine reduced Mcl-1 expression and induced apoptosis in chronic lymphocytic leukemia.

Authors:  Rong Chen; Lei Guo; Yuling Chen; Yingjun Jiang; William G Wierda; William Plunkett
Journal:  Blood       Date:  2010-10-22       Impact factor: 22.113

2.  Omacetaxine: a protein translation inhibitor for treatment of chronic myelogenous leukemia.

Authors:  Varsha Gandhi; William Plunkett; Jorge E Cortes
Journal:  Clin Cancer Res       Date:  2014-02-05       Impact factor: 12.531

3.  Cytotoxic effects of antiproliferative agents on human retinal glial cells in vitro.

Authors:  J Cai; R Wei; X Ma; H Zhu; Y Li
Journal:  Int Ophthalmol       Date:  2001       Impact factor: 2.031

Review 4.  Homoharringtonine/omacetaxine mepesuccinate: the long and winding road to food and drug administration approval.

Authors:  Hagop M Kantarjian; Susan O'Brien; Jorge Cortes
Journal:  Clin Lymphoma Myeloma Leuk       Date:  2013-06-20

Review 5.  Imatinib mesylate in combination with other chemotherapeutic agents for chronic myelogenous leukemia.

Authors:  Tetsuzo Tauchi; Kazuma Ohyashiki
Journal:  Int J Hematol       Date:  2004-06       Impact factor: 2.490

Review 6.  Accelerated Phase CML: Outcomes in Newly Diagnosed vs. Progression From Chronic Phase.

Authors:  Sudipto Mukherjee; Matt Kalaycio
Journal:  Curr Hematol Malig Rep       Date:  2016-04       Impact factor: 3.952

Review 7.  Best Practices in Chronic Myeloid Leukemia Monitoring and Management.

Authors:  Simona Soverini; Caterina De Benedittis; Manuela Mancini; Giovanni Martinelli
Journal:  Oncologist       Date:  2016-03-31

8.  Dasatinib: the emerging evidence of its potential in the treatment of chronic myeloid leukemia.

Authors:  Sonya Haslam
Journal:  Core Evid       Date:  2005-03-31

9.  mRNA translation is a therapeutic vulnerability necessary for bladder epithelial transformation.

Authors:  Sujata Jana; Rucha Deo; Rowan P Hough; Yuzhen Liu; Jessie L Horn; Jonathan L Wright; Hung-Ming Lam; Kevin R Webster; Gary G Chiang; Nahum Sonenberg; Andrew C Hsieh
Journal:  JCI Insight       Date:  2021-06-08

Review 10.  Chronic Myeloid Leukaemia in The 21st Century.

Authors:  Rachel Frazer; Alexandra E Irvine; Mary Frances McMullin
Journal:  Ulster Med J       Date:  2007-01
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