Literature DB >> 11029657

The colorectal tumour suppressor APC is present in the NMDA-receptor-PSD-95 complex in the brain.

H Yanai1, K Satoh, A Matsumine, T Akiyama.   

Abstract

BACKGROUND: The synaptic protein PSD-95/SAP90 interacts with ion channels such as the N-methyl-D-aspartate-receptor (NMDA-R) via its PDZ domain, and is involved in their clustering. Moreover, it interacts with signalling molecules and plays an important role in coupling NMDA-R to pathways that control synaptic plasticity and learning.
RESULTS: We report that PSD-95 interacts with the adenomatous polyposis coli (APC) tumour suppressor protein via its PDZ domain. Furthermore, we found that PSD-95, NMDA-R and APC are contained in the same complex in vivo. PSD-95-NMDA-R-APC association was found to require two cysteine residues conserved in the amino-terminus of PSD-95 that are known to be critical for its multimerization.
CONCLUSION: Our findings suggest that the PSD-95-NMDA-R-APC complex forms due to the multimerization of PSD-95 monomers, each of which can associate with either NMDA-R or APC. It is possible that APC is involved in the regulation of ion channel clustering and/or organization of signalling molecules.

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Year:  2000        PMID: 11029657     DOI: 10.1046/j.1365-2443.2000.00368.x

Source DB:  PubMed          Journal:  Genes Cells        ISSN: 1356-9597            Impact factor:   1.891


  14 in total

Review 1.  PDZ domains-glue and guide.

Authors:  Marco van Ham; Wiljan Hendriks
Journal:  Mol Biol Rep       Date:  2003-06       Impact factor: 2.316

2.  Laminar organization of the NMDA receptor complex within the postsynaptic density.

Authors:  J G Valtschanoff; R J Weinberg
Journal:  J Neurosci       Date:  2001-02-15       Impact factor: 6.167

3.  Activity-independent regulation of dendrite patterning by postsynaptic density protein PSD-95.

Authors:  Erik I Charych; Barbara F Akum; Joshua S Goldberg; Rebecka J Jörnsten; Christopher Rongo; James Q Zheng; Bonnie L Firestein
Journal:  J Neurosci       Date:  2006-10-04       Impact factor: 6.167

4.  APC conditional knock-out mouse is a model of infantile spasms with elevated neuronal β-catenin levels, neonatal spasms, and chronic seizures.

Authors:  Antonella Pirone; Jonathan Alexander; Lauren A Lau; David Hampton; Andrew Zayachkivsky; Amy Yee; Audrey Yee; Michele H Jacob; Chris G Dulla
Journal:  Neurobiol Dis       Date:  2016-11-13       Impact factor: 5.996

5.  To branch or not to branch: How PSD-95 regulates dendrites and spines.

Authors:  Eric S Sweet; Chia-Yi Tseng; Bonnie L Firestein
Journal:  Bioarchitecture       Date:  2011-03

6.  Kinesin Kif3b mutation reduces NMDAR subunit NR2A trafficking and causes schizophrenia-like phenotypes in mice.

Authors:  Ashwaq Hassan Alsabban; Momo Morikawa; Yosuke Tanaka; Yosuke Takei; Nobutaka Hirokawa
Journal:  EMBO J       Date:  2019-11-20       Impact factor: 11.598

7.  Adenomatous polyposis coli protein deletion leads to cognitive and autism-like disabilities.

Authors:  J L Mohn; J Alexander; A Pirone; C D Palka; S-Y Lee; L Mebane; P G Haydon; M H Jacob
Journal:  Mol Psychiatry       Date:  2014-06-17       Impact factor: 15.992

8.  Colocalization of APC and PSD-95 in the nerve fiber as well as in the post-synapse of matured neurons.

Authors:  Takanori Onouchi; Nobutaka Takamori; Takao Senda
Journal:  Med Mol Morphol       Date:  2012-09-22       Impact factor: 2.309

9.  Neuronal nicotinic synapse assembly requires the adenomatous polyposis coli tumor suppressor protein.

Authors:  Murali Krishna Temburni; Madelaine M Rosenberg; Narendra Pathak; Russell McConnell; Michele H Jacob
Journal:  J Neurosci       Date:  2004-07-28       Impact factor: 6.167

10.  Adenomatous polyposis coli plays a key role, in vivo, in coordinating assembly of the neuronal nicotinic postsynaptic complex.

Authors:  Madelaine M Rosenberg; Fang Yang; Monica Giovanni; Jesse L Mohn; Murali K Temburni; Michele H Jacob
Journal:  Mol Cell Neurosci       Date:  2008-03-04       Impact factor: 4.314

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