Literature DB >> 11005801

Animal models of spinal muscular atrophy.

U R Monani1, D D Coovert, A H Burghes.   

Abstract

Proximal spinal muscular atrophy (SMA) is the second most common autosomal recessive inherited disorder in humans. It is the most common genetic cause of infant mortality. As yet, there is no cure for this neuromuscular disorder which affects the lower motor neurons and proximal muscles of the limbs and trunk. In the last decade, significant advances have been made in understanding this disease, from linkage analysis to isolating the defective gene and identifying its protein product. This review summarizes the most recent advance in SMA research: the development of animal models of the disease, in particular mouse models of SMA. The SMA mice that we describe here present with symptoms similar to those seen in SMA patients. They promise to further the understanding of the molecular basis of this disease and demonstrate the feasibility of using the intact SMN2 gene, found in all SMA patients, as a means of treating this disorder.

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Year:  2000        PMID: 11005801     DOI: 10.1093/hmg/9.16.2451

Source DB:  PubMed          Journal:  Hum Mol Genet        ISSN: 0964-6906            Impact factor:   6.150


  42 in total

1.  Alpha-synuclein loss in spinal muscular atrophy.

Authors:  Gyula Acsadi; Xingli Li; Kelley J Murphy; Kathryn J Swoboda; Graham C Parker
Journal:  J Mol Neurosci       Date:  2010-07-17       Impact factor: 3.444

Review 2.  High throughput screening for neurodegeneration and complex disease phenotypes.

Authors:  Hemant Varma; Donald C Lo; Brent R Stockwell
Journal:  Comb Chem High Throughput Screen       Date:  2008-03       Impact factor: 1.339

Review 3.  Inflammation in ALS and SMA: sorting out the good from the evil.

Authors:  Dimitra Papadimitriou; Virginia Le Verche; Arnaud Jacquier; Burcin Ikiz; Serge Przedborski; Diane B Re
Journal:  Neurobiol Dis       Date:  2009-10-13       Impact factor: 5.996

4.  The spinal muscular atrophy mouse model, SMAΔ7, displays altered axonal transport without global neurofilament alterations.

Authors:  Jeffrey M Dale; Hailian Shen; Devin M Barry; Virginia B Garcia; Ferrill F Rose; Christian L Lorson; Michael L Garcia
Journal:  Acta Neuropathol       Date:  2011-06-17       Impact factor: 17.088

5.  An approximately 140-kb deletion associated with feline spinal muscular atrophy implies an essential LIX1 function for motor neuron survival.

Authors:  John C Fyfe; Marilyn Menotti-Raymond; Victor A David; Lars Brichta; Alejandro A Schäffer; Richa Agarwala; William J Murphy; William J Wedemeyer; Brittany L Gregory; Bethany G Buzzell; Meghan C Drummond; Brunhilde Wirth; Stephen J O'Brien
Journal:  Genome Res       Date:  2006-08-09       Impact factor: 9.043

Review 6.  Spinal muscular atrophy.

Authors:  Susan T Iannaccone; Stephen A Smith; Louise R Simard
Journal:  Curr Neurol Neurosci Rep       Date:  2004-01       Impact factor: 5.081

7.  Mutant glycyl-tRNA synthetase (Gars) ameliorates SOD1(G93A) motor neuron degeneration phenotype but has little affect on Loa dynein heavy chain mutant mice.

Authors:  Gareth T Banks; Virginie Bros-Facer; Hazel P Williams; Ruth Chia; Francesca Achilli; J Barney Bryson; Linda Greensmith; Elizabeth M C Fisher
Journal:  PLoS One       Date:  2009-07-13       Impact factor: 3.240

8.  Neurotransmitter release in motor nerve terminals of a mouse model of mild spinal muscular atrophy.

Authors:  Rocío Ruiz; Lucía Tabares
Journal:  J Anat       Date:  2013-03-13       Impact factor: 2.610

9.  A positive modifier of spinal muscular atrophy in the SMN2 gene.

Authors:  Thomas W Prior; Adrian R Krainer; Yimin Hua; Kathryn J Swoboda; Pamela C Snyder; Scott J Bridgeman; Arthur H M Burghes; John T Kissel
Journal:  Am J Hum Genet       Date:  2009-08-27       Impact factor: 11.025

Review 10.  Studying human disease using human neurons.

Authors:  Tim Ahfeldt; Nadia K Litterman; Lee L Rubin
Journal:  Brain Res       Date:  2016-04-06       Impact factor: 3.252

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