Literature DB >> 10992554

Role of leukotrienes in bronchial hyperresponsiveness and cellular responses in airways.

A R Leff1.   

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Year:  2000        PMID： 10992554      PMCID： PMC1765960          DOI： 10.1136/thorax.55.suppl_2.s32

Source DB:  PubMed          Journal:  Thorax        ISSN： 0040-6376            Impact factor:   9.139


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  25 in total

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Review 2.  The eosinophil and bronchial asthma: current understanding.

Authors:  G J Gleich
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3.  Endothelial leukocyte adhesion molecule 1: an inducible receptor for neutrophils related to complement regulatory proteins and lectins.

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4.  Specific inhibition of leukotriene B4-induced neutrophil activation by LY223982.

Authors:  W T Jackson; R J Boyd; L L Froelich; B E Mallett; D M Gapinski
Journal:  J Pharmacol Exp Ther       Date:  1992-12       Impact factor: 4.030

5.  ONO-4057, a novel, orally active leukotriene B4 antagonist: effects on LTB4-induced neutrophil functions.

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6.  Effects of activated eosinophils cultured from human umbilical cord blood on guinea pig trachealis.

Authors:  K J Hamann; M E Strek; S L Baranowski; N M Munoz; F S Williams; S R White; A Vita; A R Leff
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7.  Activation of tracheal smooth muscle responsiveness by fMLP-treated HL-60 cells and neutrophils.

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8.  Augmentation of stimulated eosinophil degranulation by VLA-4 (CD49d)-mediated adhesion to fibronectin.

Authors:  S P Neeley; K J Hamann; T L Dowling; K T McAllister; S R White; A R Leff
Journal:  Am J Respir Cell Mol Biol       Date:  1994-08       Impact factor: 6.914

9.  Augmented muscarinic responsiveness caused by 5-lipoxygenase products secreted from alveolar macrophages in isolated-perfused rat lung.

Authors:  P Padrid; R Wolf; N M Munoz; S Spaethe; T Finucane; J Solway; A R Leff
Journal:  Am Rev Respir Dis       Date:  1993-06

10.  Intercellular adhesion molecule-1 (ICAM-1) in the pathogenesis of asthma.

Authors:  C D Wegner; R H Gundel; P Reilly; N Haynes; L G Letts; R Rothlein
Journal:  Science       Date:  1990-01-26       Impact factor: 47.728
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2.  Dynamic model of eicosanoid production with special reference to non-steroidal anti-inflammatory drug-triggered hypersensitivity.

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