Literature DB >> 10980881

The myocardial matrix and the development and progression of ventricular remodeling.

J D Sackner-Bernstein1.   

Abstract

Our conceptual framework of chronic heart failure is based upon the neurohormonal model. In this construct, neurohormonal systems that provide short-term homeostasis remain activated after a myocardial injury, producing progressive ventricular dysfunction and worsening heart failure. However, this model fails to explain several important clinical phenomena, that can be explained by an expanded model of heart failure that focuses on myocardial matrix events as the triggers for disease progression. This model embraces the neurohormonal model and integrates the roles of the immune system and the myocardial fibroblast within the matrix to more fully describe the initiation and progression of the disease.

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Year:  2000        PMID： 10980881     DOI： 10.1007/s11886-000-0007-4

Source DB:  PubMed          Journal:  Curr Cardiol Rep        ISSN： 1523-3782            Impact factor:   3.955


  88 in total

Review 1.  Regulation of cardiovascular collagen synthesis by mechanical load.

Authors:  J E Bishop; G Lindahl
Journal:  Cardiovasc Res       Date:  1999-04       Impact factor: 10.787

Review 2.  The role of nitric oxide and cytokines in heart failure.

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Journal:  Coron Artery Dis       Date:  1997-06       Impact factor: 1.439

3.  Cultured myofibroblasts generate angiotensin peptides de novo.

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Journal:  J Mol Cell Cardiol       Date:  1997-05       Impact factor: 5.000

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Authors:  H N Sabbah; V G Sharov
Journal:  Prog Cardiovasc Dis       Date:  1998 May-Jun       Impact factor: 8.194

5.  Differential gene expression of extracellular matrix components in dilated cardiomyopathy.

Authors:  S C Tyagi; S Kumar; D J Voelker; H K Reddy; J S Janicki; J J Curtis
Journal:  J Cell Biochem       Date:  1996-11-01       Impact factor: 4.429

6.  alpha1-adrenergic receptor subtypes in human peripheral blood lymphocytes.

Authors:  A Ricci; E Bronzetti; A Conterno; S Greco; P Mulatero; M Schena; D Schiavone; S K Tayebati; F Veglio; F Amenta
Journal:  Hypertension       Date:  1999-02       Impact factor: 10.190

7.  Stimulation of adult rat ventricular myocyte protein synthesis and phosphoinositide hydrolysis by the endothelins.

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Journal:  Biochim Biophys Acta       Date:  1993-02-17

8.  Ischemia/reperfusion-induced granulocyte influx is a multistep process mediated by mast cells.

Authors:  S Kanwar; P Kubes
Journal:  Microcirculation       Date:  1994-10       Impact factor: 2.628

9.  Induction of interleukin-6 synthesis in the myocardium. Potential role in postreperfusion inflammatory injury.

Authors:  G L Kukielka; C W Smith; A M Manning; K A Youker; L H Michael; M L Entman
Journal:  Circulation       Date:  1995-10-01       Impact factor: 29.690

10.  Cytokine gene expression after myocardial infarction in rat hearts: possible implication in left ventricular remodeling.

Authors:  K Ono; A Matsumori; T Shioi; Y Furukawa; S Sasayama
Journal:  Circulation       Date:  1998-07-14       Impact factor: 29.690
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  7 in total

Review 1.  Adverse Remodeling and Reverse Remodeling After Myocardial Infarction.

Authors:  Ankeet S Bhatt; Andrew P Ambrosy; Eric J Velazquez
Journal:  Curr Cardiol Rep       Date:  2017-08       Impact factor: 2.931

Review 2.  Novel and potential future biomarkers for assessment of the severity and prognosis of chronic heart failure : a clinical review.

Authors:  David R Buvat de Virginy
Journal:  Heart Fail Rev       Date:  2006-12       Impact factor: 4.214

3.  NT-proBNP, C-reactive protein and soluble uPAR in a bi-ethnic male population: the SAfrEIC study.

Authors:  Ruan Kruger; Rudolph Schutte; Hugo W Huisman; Peter Hindersson; Michael H Olsen; Jesper Eugen-Olsen; Aletta E Schutte
Journal:  PLoS One       Date:  2013-03-13       Impact factor: 3.240

4.  Extracellular matrix biomarker, fibulin-1, is closely related to NT-proBNP and soluble urokinase plasminogen activator receptor in patients with aortic valve stenosis (the SEAS study).

Authors:  Ruan Kruger; Lars M Rasmussen; William S Argraves; Jesper Eugen-Olsen; Olav W Nielsen; Adam Blyme; Ronnie Willenheimer; Kristian Wachtell; Michael H Olsen
Journal:  PLoS One       Date:  2014-07-11       Impact factor: 3.240

5.  Fibrosis of extracellular matrix is related to the duration of the disease but is unrelated to the dynamics of collagen metabolism in dilated cardiomyopathy.

Authors:  Paweł Rubiś; Sylwia Wiśniowska-Śmialek; Ewa Wypasek; Barbara Biernacka-Fijalkowska; Lucyna Rudnicka-Sosin; Ewa Dziewiecka; Patrycja Faltyn; Lusine Khachatryan; Aleksandra Karabinowska; Artur Kozanecki; Lidia Tomkiewicz-Pająk; Piotr Podolec
Journal:  Inflamm Res       Date:  2016-08-11       Impact factor: 4.575

6.  The relationship between myocardial fibrosis and myocardial microRNAs in dilated cardiomyopathy: A link between mir-133a and cardiovascular events.

Authors:  Paweł Rubiś; Justyna Totoń-Żurańska; Sylwia Wiśniowska-Śmiałek; Ewa Dziewięcka; Maria Kołton-Wróż; Paweł Wołkow; Ewelina Pitera; Lucyna Rudnicka-Sosin; Ann C Garlitski; Andrzej Gackowski; Piotr Podolec
Journal:  J Cell Mol Med       Date:  2018-01-29       Impact factor: 5.310

7.  Modulation of miR-10a-mediated TGF-β1/Smads signaling affects atrial fibrillation-induced cardiac fibrosis and cardiac fibroblast proliferation.

Authors:  Peng-Fei Li; Rong-Hua He; Shao-Bo Shi; Rui Li; Qiong-Tao Wang; Guo-Tao Rao; Bo Yang
Journal:  Biosci Rep       Date:  2019-02-08       Impact factor: 3.840
  7 in total

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