Literature DB >> 10971313

Intravenous diltiazem and CYP3A-mediated metabolism.

A L Masica1, N E Azie, D C Brater, S D Hall, D R Jones.   

Abstract

AIMS: To study whether intravenous diltiazem, a calcium channel blocker commonly prescribed for hypertension and stable angina, is an inhibitor of the CYP3A enzymes by using oral lovastatin, an HMG Co-A reductase inhibitor, as a substrate.
METHODS: Ten healthy volunteers were studied in a randomized two-way crossover design. The two arms were 1) administration of a 20 mg dosage of lovastatin orally and 2) administration of a 20 mg dosage of lovastatin orally 1 h after an intravenous loading dosage and constant infusion of diltiazem. Blood samples were collected up to 25 h in order to quantify lovastatin and diltiazem concentrations in the separated serum. Lovastatin and diltiazem concentrations were quantified by GC-MS and h.p.l.c., respectively.
RESULTS: Intravenous diltiazem did not significantly affect the oral AUC, Cmax, t(1/2), or tmax of lovastatin.
CONCLUSIONS: These data suggest that the interaction of lovastatin with diltiazem does not occur systemically and is primarily a first-pass effect. Thus, drug interactions with diltiazem may become evident when a patient is moved from intravenous to oral dosing.

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Year:  2000        PMID: 10971313      PMCID: PMC2014983          DOI: 10.1046/j.1365-2125.2000.00249.x

Source DB:  PubMed          Journal:  Br J Clin Pharmacol        ISSN: 0306-5251            Impact factor:   4.335


  22 in total

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8.  Diltiazem treatment impairs hepatic drug oxidation: studies of antipyrine.

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9.  Identification of rifampin-inducible P450IIIA4 (CYP3A4) in human small bowel enterocytes.

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10.  Acute hemodynamic and electrophysiologic effects and safety of high-dose intravenous diltiazem in patients receiving metoprolol.

Authors:  A C Wiesfeld; W J Remme; M P Look; D A Kruijssen; D C van Hoogenhuyze
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