Literature DB >> 10951621

[Resistance of Bcl-2 adenovirus vector to HepG(2) cell apoptosis induced by ethanol].

B Zhang1, D Zhang, H Ren.   

Abstract

OBJECTIVE: To evaluate the role of Bcl-2 family proteins in hepatocytic apoptosis caused by ethanol.
METHODS: We induced hepatocytic apoptosis in HepG(2) cells with ethanol and detected the apoptosis with TUNEL assay. Bcl-2, Bax, and Bak protein expression in the hepatocytes was determined by immunohistochemistry method. We also observed the expression of Bcl-2 protein in HepG(2) cells infected with Bcl-2 adenovirus vector and its protection against hepatocytic apoptosis caused by ethanol.
RESULTS: No obvious cytotoxicity was noted in 0.2% and 1% ethanol treatment group, while it developed in 3% ethanol treatment group and accompanied by a marked expression of Bax, Bak proteins.
CONCLUSION: The overexpression of Bax, Bak proteins may play a role in HepG(2) cell apoptosis induced by ethanol and can be blocked effectively by Bcl-2 adenovirus vector.

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Year:  2000        PMID: 10951621

Source DB:  PubMed          Journal:  Zhonghua Gan Zang Bing Za Zhi        ISSN: 1007-3418


  3 in total

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Journal:  Bioorg Med Chem       Date:  2011-08-26       Impact factor: 3.641

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Authors:  Jun Wu; Ming-Liang Cheng; Guo-Hao Zhang; Rong-Wei Zhai; Neng-Hui Huang; Cheng-Xiu Li; Tian-Yong Luo; Shuang Lu; Zhi-Qin Yu; Yu-Mei Yao; Ying-Ying Zhang; Lan-Zhen Ren; Lan Ye; Ling Li; Hui-Na Zhang
Journal:  World J Gastroenterol       Date:  2002-06       Impact factor: 5.742

3.  Ethanol promotes T cell apoptosis through the mitochondrial pathway.

Authors:  Aditi A Kapasi; Geeta Patel; Anuj Goenka; Nilay Nahar; Neeraj Modi; Madhu Bhaskaran; Krishna Reddy; Nicholas Franki; Jaimita Patel; Pravin C Singhal
Journal:  Immunology       Date:  2003-03       Impact factor: 7.397

  3 in total

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