Literature DB >> 10931408

Predictors of coronary artery lesions after intravenous gamma-globulin treatment in Kawasaki disease.

M Mori1, T Imagawa, K Yasui, A Kanaya, S Yokota.   

Abstract

OBJECTIVE: We evaluated the efficacy of intravenous gamma-globulin (IVGG) administration for children with Kawasaki disease to establish whether additional, more advanced therapy is needed in intractable cases. STUDY
DESIGN: A total of 193 children with Kawasaki disease were studied retrospectively. Patients were admitted 3 to 7 days after the onset of the disease, and IVGG was administered. Laboratory measurements including white blood cell (WBC), neutrophil, and platelet counts and C-reactive protein (CRP) and albumin concentrations were determined before and 2 to 3 days after IVGG treatment. The progression of coronary artery lesions (CALs) was monitored by serial echocardiography until 30 days after treatment.
RESULTS: Of 193 children, 24 (12.2 %) had CALs including transient dilatation. In contrast to the other measurements, the WBC count increased in 21 of 24 (87.5%) children with CALs after IVGG therapy. The patients with increased neutrophil count and CRP concentration after IVGG therapy also had CAL formation at a high rate (78.3% and 66.7%, respectively). Among children with normal coronary arteries, elevations of the WBC and neutrophil counts and CRP concentration were observed after IVGG therapy in only 3, 6, and 8 patients, respectively (specificity: 98.2%, 97.0%, and 95.3%, respectively). Furthermore, multiple logistic regression indicated that these variables were useful predictors of CALs in KD.
CONCLUSION: Though the introduction of IVGG therapy has improved the prognosis of Kawasaki disease, approximately 10% of patients still develop CALs. The need for more aggressive therapy in IVGG-resistant cases can be recognized early by increases in the WBC and neutrophil counts and serum CRP concentration after IVGG administration.

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Year:  2000        PMID: 10931408     DOI: 10.1067/mpd.2000.107890

Source DB:  PubMed          Journal:  J Pediatr        ISSN: 0022-3476            Impact factor:   4.406


  33 in total

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2.  Plasma exchange for refractory Kawasaki disease.

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4.  Common variants in the CRP promoter are associated with a high C-reactive protein level in Kawasaki disease.

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5.  Extensive coronary aneurysms with thrombosis in resistant Kawasaki disease.

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7.  Association of the matrix metalloproteinase-3 (-439C/G) promoter polymorphism with Kawasaki disease in Korean children.

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10.  The CCR5 (-2135C/T) polymorphism may be associated with the development of Kawasaki disease in Korean children.

Authors:  Won Kyoung Jhang; Mi-Jin Kang; Hyun-Seung Jin; Jinho Yu; Byoung-ju Kim; Bong Seong Kim; Jong-Keuk Lee; Eul-Ju Seo; Han-Wook Yoo; In Sook Park; Young Mi Hong; Soo-Jong Hong
Journal:  J Clin Immunol       Date:  2008-07-15       Impact factor: 8.317

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