Literature DB >> 10920397

Selective nuclear export of viral mRNAs in influenza-virus-infected cells.

Z Chen1, R M Krug.   

Abstract

The NS1A protein of influenza A virus specifically inhibits the cellular machinery that processes the 3' ends of cellular pre-mRNAs by targeting two of the essential proteins of this machinery. Because the virus does not use this cellular machinery to synthesize the 3' poly(A) ends of viral mRNA, the nuclear export of cellular but not viral mRNAs is selectively inhibited.

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Year:  2000        PMID： 10920397     DOI： 10.1016/s0966-842x(00)01794-7

Source DB:  PubMed          Journal:  Trends Microbiol        ISSN： 0966-842X            Impact factor:   17.079


  24 in total

1.  Genetic analysis of influenza virus NS1 gene: a temperature-sensitive mutant shows defective formation of virus particles.

Authors:  Urtzi Garaigorta; Ana M Falcón; Juan Ortín
Journal:  J Virol       Date:  2005-12       Impact factor: 5.103

2.  Influenza B virus NS1 protein inhibits conjugation of the interferon (IFN)-induced ubiquitin-like ISG15 protein.

Authors:  W Yuan; R M Krug
Journal:  EMBO J       Date:  2001-02-01       Impact factor: 11.598

3.  Functional dissection of the zinc finger and flanking domains of the Yth1 cleavage/polyadenylation factor.

Authors:  Yoko Tacahashi; Steffen Helmling; Claire L Moore
Journal:  Nucleic Acids Res       Date:  2003-03-15       Impact factor: 16.971

4.  Rna15 interaction with the A-rich yeast polyadenylation signal is an essential step in mRNA 3'-end formation.

Authors:  S Gross; C L Moore
Journal:  Mol Cell Biol       Date:  2001-12       Impact factor: 4.272

Review 5.  Cellular networks involved in the influenza virus life cycle.

Authors:  Tokiko Watanabe; Shinji Watanabe; Yoshihiro Kawaoka
Journal:  Cell Host Microbe       Date:  2010-06-25       Impact factor: 21.023

6.  Energetic cost of building a virus.

Authors:  Gita Mahmoudabadi; Ron Milo; Rob Phillips
Journal:  Proc Natl Acad Sci U S A       Date:  2017-05-16       Impact factor: 11.205

7.  Distribution and dynamics of transcription-associated proteins during parvovirus infection.

Authors:  Teemu O Ihalainen; Sami F Willman; Einari A Niskanen; Outi Paloheimo; Hanna Smolander; Juha P Laurila; Minna U Kaikkonen; Maija Vihinen-Ranta
Journal:  J Virol       Date:  2012-10-03       Impact factor: 5.103

8.  Bidirectional increase in permeability of nuclear envelope upon poliovirus infection and accompanying alterations of nuclear pores.

Authors:  George A Belov; Peter V Lidsky; Olga V Mikitas; Denise Egger; Konstantin A Lukyanov; Kurt Bienz; Vadim I Agol
Journal:  J Virol       Date:  2004-09       Impact factor: 5.103

9.  The N- and C-terminal domains of the NS1 protein of influenza B virus can independently inhibit IRF-3 and beta interferon promoter activation.

Authors:  Nicola R Donelan; Bianca Dauber; Xiuyan Wang; Christopher F Basler; Thorsten Wolff; Adolfo García-Sastre
Journal:  J Virol       Date:  2004-11       Impact factor: 5.103

10.  Human influenza viruses activate an interferon-independent transcription of cellular antiviral genes: outcome with influenza A virus is unique.

Authors:  Mee-Jung Kim; Anita Ghate Latham; Robert M Krug
Journal:  Proc Natl Acad Sci U S A       Date:  2002-07-11       Impact factor: 11.205
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