Literature DB >> 10916677

Chronic non-spherocytic haemolytic disorders associated with glucose-6-phosphate dehydrogenase variants.

G Fiorelli1, F Martinez di Montemuros, M D Cappellini.   

Abstract

Glucose-6-phosphate dehydrogenase (G6PD) deficiency is the most common human enzyme defect, being present in over 400 million people world wide. In a small number of cases, G6PD deficiency can lead to mild-to-severe chronic haemolysis, which is further exacerbated by oxidative stress. Such G6PD variants have been described all over the world and are responsible for chronic non-spherocytic haemolytic anaemia (CNSHA). To date 61 G6PD molecular variants associated with CNSHA have been identified, only some of which can cause the severe reduction in stability of the red blood cell enzyme. The distribution of the different mutations shows a predominance of small mutational events, and many have been found repeatedly in different parts of the world. By revisiting the 61 class I variants described so far, we can observe that a low inhibition constant (Ki) for NADPH, a higher Km for substrates and a reduced thermostability are common.

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Year:  2000        PMID: 10916677     DOI: 10.1053/beha.1999.0056

Source DB:  PubMed          Journal:  Baillieres Best Pract Res Clin Haematol


  13 in total

1.  Life and Death of Glucose-6-Phosphate Dehydrogenase (G6PD) Deficient Erythrocytes - Role of Redox Stress and Band 3 Modifications.

Authors:  Paolo Arese; Valentina Gallo; Antonella Pantaleo; Franco Turrini
Journal:  Transfus Med Hemother       Date:  2012-09-17       Impact factor: 3.747

2.  Molecular Characterization of G6PD Deficiency: Report of Three Novel G6PD Variants.

Authors:  Arun Kumar Arunachalam; S Sumithra; Madhavi Maddali; N A Fouzia; Aby Abraham; Biju George; Eunice S Edison
Journal:  Indian J Hematol Blood Transfus       Date:  2019-10-16       Impact factor: 0.900

3.  In silico model-driven assessment of the effects of single nucleotide polymorphisms (SNPs) on human red blood cell metabolism.

Authors:  Neema Jamshidi; Sharon J Wiback; Bernhard Ø Palsson B
Journal:  Genome Res       Date:  2002-11       Impact factor: 9.043

4.  A novel G6PD mutation leading to chronic hemolytic anemia.

Authors:  Jenny McDade; Tatiana Abramova; Nicole Mortier; Thad Howard; Russell E Ware
Journal:  Pediatr Blood Cancer       Date:  2008-12       Impact factor: 3.167

5.  Relating mutant genotype to phenotype via quantitative behavior of the NADPH redox cycle in human erythrocytes.

Authors:  Pedro M B M Coelho; Armindo Salvador; Michael A Savageau
Journal:  PLoS One       Date:  2010-09-28       Impact factor: 3.240

6.  Predicting the Kinetic Properties Associated with Redox Imbalance after Oxidative Crisis in G6PD-Deficient Erythrocytes: A Simulation Study.

Authors:  Hanae Shimo; Taiko Nishino; Masaru Tomita
Journal:  Adv Hematol       Date:  2011-09-28

7.  Dynamic simulation of red blood cell metabolism and its application to the analysis of a pathological condition.

Authors:  Yoichi Nakayama; Ayako Kinoshita; Masaru Tomita
Journal:  Theor Biol Med Model       Date:  2005-05-09       Impact factor: 2.432

Review 8.  Diagnostic approaches for inherited hemolytic anemia in the genetic era.

Authors:  Yonggoo Kim; Joonhong Park; Myungshin Kim
Journal:  Blood Res       Date:  2017-06-22

9.  Using in silico models to simulate dual perturbation experiments: procedure development and interpretation of outcomes.

Authors:  Neema Jamshidi; Bernhard O Palsson
Journal:  BMC Syst Biol       Date:  2009-04-30

10.  Safety of 8-aminoquinolines given to people with G6PD deficiency: protocol for systematic review of prospective studies.

Authors:  Olalekan A Uthman; Rachel Saunders; David Sinclair; Patricia Graves; Hellen Gelband; Aileen Clarke; Paul Garner
Journal:  BMJ Open       Date:  2014-05-14       Impact factor: 2.692

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