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Literature DB >> 10908222

Left ventricular hypertrophy: pathogenesis, detection, and prognosis.

B H Lorell¹, B A Carabello.

Abstract

Entities: Disease

Keywords: NASA Discipline Cardiopulmonary; Non-NASA Center

Mesh：

Year: 2000 PMID： 10908222 DOI： 10.1161/01.cir.102.4.470

Source DB: PubMed Journal: Circulation ISSN： 0009-7322 Impact factor: 29.690

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Cited

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294 in total

1. Resveratrol prevents pathological but not physiological cardiac hypertrophy.

Authors: Vernon W Dolinsky; Carrie-Lynn M Soltys; Kyle J Rogan; Anita Y M Chan; Jeevan Nagendran; Shaohua Wang; Jason R B Dyck
Journal: J Mol Med (Berl) Date: 2014-11-15 Impact factor: 4.599

2. Interaction between NFκB and NFAT coordinates cardiac hypertrophy and pathological remodeling.

Authors: Qinghang Liu; Yi Chen; Mannix Auger-Messier; Jeffery D Molkentin
Journal: Circ Res Date: 2012-03-08 Impact factor: 17.367

3. A friend within the heart: natriuretic peptide receptor signaling.

Authors: Jeffery D Molkentin
Journal: J Clin Invest Date: 2003-05 Impact factor: 14.808

Review 4. Gene silencing in DNA damage repair.

Authors: H Soejima; K Joh; T Mukai
Journal: Cell Mol Life Sci Date: 2004-09 Impact factor: 9.261

Review 5. Cardiac remodeling at the population level--risk factors, screening, and outcomes.

Authors: Ola Gjesdal; David A Bluemke; Joao A Lima
Journal: Nat Rev Cardiol Date: 2011-10-25 Impact factor: 32.419

6. Effects of increased preload on the force-frequency response and contractile kinetics in early stages of cardiac muscle hypertrophy.

Authors: Kaylan M Haizlip; Tepmanas Bupha-Intr; Brandon J Biesiadecki; Paul M L Janssen
Journal: Am J Physiol Heart Circ Physiol Date: 2012-03-30 Impact factor: 4.733

Left ventricular hypertrophy: pathogenesis, detection, and prognosis.

1. Resveratrol prevents pathological but not physiological cardiac hypertrophy.

2. Interaction between NFκB and NFAT coordinates cardiac hypertrophy and pathological remodeling.

3. A friend within the heart: natriuretic peptide receptor signaling.

Review 4. Gene silencing in DNA damage repair.

Review 5. Cardiac remodeling at the population level--risk factors, screening, and outcomes.

6. Effects of increased preload on the force-frequency response and contractile kinetics in early stages of cardiac muscle hypertrophy.

7. Matrix elasticity regulates the optimal cardiac myocyte shape for contractility.

8. c-Jun N-terminal kinases (JNK) antagonize cardiac growth through cross-talk with calcineurin-NFAT signaling.

Review 9. Role of ubiquitin-proteasome system (UPS) in left ventricular hypertrophy (LVH).

Review 10. Regulation of cardiac hypertrophy and remodeling through the dual-specificity MAPK phosphatases (DUSPs).