| Literature DB >> 10881267 |
T Ohno1, M Bando, H Nagura, K Ishii, H Yamanouchi.
Abstract
The authors report a patient of pure apraxic agraphia with normal praxis due to left thalamic infarction. 15O-gas-PET showed reduced oxygen metabolism in the left thalamus and the left dorsolateral premotor area, while MRI and 11C-fulumazenil-PET showed no remarkable lesions in the frontal cortex. The patient's word imaging remained normal. The authors hypothesize that thalamic destruction causes pure apraxic agraphia by exerting a remote effect on left dorsolateral premotor area and blocking somewhere between graphemic area and motor programming.Entities:
Mesh:
Year: 2000 PMID: 10881267 DOI: 10.1212/wnl.54.12.2336
Source DB: PubMed Journal: Neurology ISSN: 0028-3878 Impact factor: 9.910