Literature DB >> 10879622

Early rather than delayed administration of lisinopril protects the heart after myocardial infarction in rats.

L A Zornoff1, B B Matsubara, L S Matsubara, S A Paiva, J Spadaro.   

Abstract

BACKGROUND: ACE inhibitors have shown beneficial results in several studies after myocardial infarction (MI). However, these studies have shown conflicting results about the ideal starting time of the ACE inhibitors administration after MI and the importance of infarct size.
OBJECTIVES: This study was designed to assess the long-term effects of lisinopril on mortality, cardiac function, and ventricular fibrosis after MI, in rats.
METHODS: Lisinopril (20 mg/kg/day) was given on day 1 or 21 days after coronary occlusion in small or large infarctions.
RESULTS: The mortality rate was reduced by 39 % in early treatment and 30 % in delayed treatment in comparison to the untreated rats. Early treatment reduced cardiac dysfunction in small MIs; however, delayed treatment did not. No statistical difference was observed among the groups for large MIs. No statistical difference was observed among the groups with large or small MIs on myocardial hydroxyproline concentration.
CONCLUSIONS: Both early and delayed treatments with lisinopril increased survival. Treatment exerts no marked effects on fibrosis; early treatment has exerted beneficial influences on cardiac function whereas delayed treatment had no consistent effects. The protective effect of lisinopril is detectable only in small (< 40 % of LV) MIs.

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Year:  2000        PMID: 10879622     DOI: 10.1007/s003950050183

Source DB:  PubMed          Journal:  Basic Res Cardiol        ISSN: 0300-8428            Impact factor:   17.165


  8 in total

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Authors:  Bertha F Polegato; Marcos F Minicucci; Paula S Azevedo; Andréa F Gonçalves; Aline F Lima; Paula F Martinez; Marina P Okoshi; Katashi Okoshi; Sergio A R Paiva; Leonardo A M Zornoff
Journal:  Arq Bras Cardiol       Date:  2016-01-26       Impact factor: 2.000

4.  Early echocardiographic predictors of increased left ventricular end-diastolic pressure three months after myocardial infarction in rats.

Authors:  Paula S Azevedo; Bertha F Polegato; Marcos F Minicucci; Stephan M Pio; Igor A Silva; Priscila P Santos; Katashi Okoshi; Sergio A R Paiva; Leonardo A M Zornoff
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5.  The role of oxidative stress and lipid peroxidation in ventricular remodeling induced by tobacco smoke exposure after myocardial infarction.

Authors:  Daniella R Duarte; Marcos F Minicucci; Paula S Azevedo; Beatriz B Matsubara; Luiz S Matsubara; Ethel L Novelli; Sergio A R Paiva; Leonardo A M Zornoff
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6.  Periostin as a modulator of chronic cardiac remodeling after myocardial infarction.

Authors:  Marcos F Minicucci; Priscila P dos Santos; Bruna P M Rafacho; Andréa F Gonçalves; Lidiane P Ardisson; Diego F Batista; Paula S Azevedo; Bertha F Polegato; Katashi Okoshi; Elenize J Pereira; Sergio A R Paiva; Leonardo A M Zornoff
Journal:  Clinics (Sao Paulo)       Date:  2013-10       Impact factor: 2.365

7.  Mechanisms involved in the beneficial effects of spironolactone after myocardial infarction.

Authors:  Marcos F Minicucci; Priscila P dos Santos; Bruna P M Rafacho; Andrea F Gonçalves; Renata A C Silva; Fernanda Chiuso-Minicucci; Paula S Azevedo; Bertha F Polegato; Katashi Okoshi; Elenize J Pereira; Sergio A R Paiva; Leonardo A M Zornoff
Journal:  PLoS One       Date:  2013-09-30       Impact factor: 3.240

8.  Angiotensin-converting-enzyme inhibitor prevents skeletal muscle fibrosis in myocardial infarction mice.

Authors:  Naoya Kakutani; Shingo Takada; Hideo Nambu; Junichi Matsumoto; Takaaki Furihata; Takashi Yokota; Arata Fukushima; Shintaro Kinugawa
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  8 in total

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