Literature DB >> 10877738

3-year suppression of HIV viremia with indinavir, zidovudine, and lamivudine.

R M Gulick1, J W Mellors, D Havlir, J J Eron, A Meibohm, J H Condra, F T Valentine, D McMahon, C Gonzalez, L Jonas, E A Emini, J A Chodakewitz, R Isaacs, D D Richman.   

Abstract

BACKGROUND: Antiretroviral regimens containing HIV protease inhibitors suppress viremia in HIV-infected patients, but the durability of this effect is not known.
OBJECTIVE: To describe the 3-year follow-up of patients randomly assigned to receive indinavir, zidovudine, and lamivudine in an ongoing clinical trial.
DESIGN: Open-label extension of a randomized, double-blind study.
SETTING: Four clinical research units. PATIENTS: 33 HIV-infected, zidovudine-experienced patients with serum HIV RNA levels of at least 20,000 copies/mL and CD4 counts ranging from 50 to 400 cells/mm3. INTERVENTION: Indinavir, zidovudine, and lamivudine. MEASUREMENTS: Safety assessments, HIV RNA levels, CD4 cell counts, and genotypic analyses.
RESULTS: After 3 years of follow-up, 21 of 31 contributing patients (68% [95% CI, 49% to 83%]) had serum viral load levels less than 500 copies/mL. Twenty of 31 (65% [CI, 45% to 80%]) had levels less than 50 copies/mL. The median increase in CD4 count from baseline was 230 cells/mm3 (interquartile range, 150 to 316 cells/mm3). Nephrolithiasis occurred in 12 of 33 patients (36%).
CONCLUSION: A three-drug regimen of indinavir, zidovudine, and lamivudine suppressed viremia in two thirds of patients for at least 3 years.

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Year:  2000        PMID: 10877738     DOI: 10.7326/0003-4819-133-1-200007040-00007

Source DB:  PubMed          Journal:  Ann Intern Med        ISSN: 0003-4819            Impact factor:   25.391


  36 in total

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Authors:  Rieneke M E van Praag; Elisabeth C M van Weert; Rolf P G van Heeswijk; Xiao-Jian Zhou; Jean-Pierre Sommadossi; Suzanne Jurriaans; Joep M A Lange; Richard M W Hoetelmans; Jan M Prins
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2.  Induction of apoptosis by a nonnucleoside human immunodeficiency virus type 1 reverse transcriptase inhibitor.

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3.  Pharmacokinetics of indinavir combined with low-dose ritonavir in human immunodeficiency virus type 1-infected children.

Authors:  A S Bergshoeff; P L A Fraaij; A M C van Rossum; G Verweel; L H Wynne; G A Winchell; R Y Leavitt; B-Y T Nguyen; R de Groot; D M Burger
Journal:  Antimicrob Agents Chemother       Date:  2004-05       Impact factor: 5.191

4.  Nine crystal structures determine the substrate envelope of the MDR HIV-1 protease.

Authors:  Zhigang Liu; Yong Wang; Joseph Brunzelle; Iulia A Kovari; Ladislau C Kovari
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Review 6.  Beyond one pill, once daily: current challenges of antiretroviral therapy management in the United States.

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7.  Design, synthesis, and biological and structural evaluations of novel HIV-1 protease inhibitors to combat drug resistance.

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8.  Productive infection maintains a dynamic steady state of residual viremia in human immunodeficiency virus type 1-infected persons treated with suppressive antiretroviral therapy for five years.

Authors:  Diane V Havlir; Matthew C Strain; Mario Clerici; Caroline Ignacio; Daria Trabattoni; Pasquale Ferrante; Joseph K Wong
Journal:  J Virol       Date:  2003-10       Impact factor: 5.103

9.  Effectiveness of nonpeptide clinical inhibitor TMC-114 on HIV-1 protease with highly drug resistant mutations D30N, I50V, and L90M.

Authors:  Andrey Yu Kovalevsky; Yunfeng Tie; Fengling Liu; Peter I Boross; Yuan-Fang Wang; Sofiya Leshchenko; Arun K Ghosh; Robert W Harrison; Irene T Weber
Journal:  J Med Chem       Date:  2006-02-23       Impact factor: 7.446

10.  Indinavir/low-dose ritonavir containing HAART in HIV-1 infected children has potent antiretroviral activity, but is associated with side effects and frequent discontinuation of treatment.

Authors:  P L A Fraaij; G Verweel; A M C van Rossum; N G Hartwig; D M Burger; R de Groot
Journal:  Infection       Date:  2007-06       Impact factor: 3.553

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