Literature DB >> 10755538

Incidence, progression and functional significance of cardiac allograft vasculopathy after heart transplantation.

B K Julius1, C H Attenhofer Jost, G Sütsch, H P Brunner, A Kuenzli, P R Vogt, M Turina, O M Hess, W Kiowski.   

Abstract

BACKGROUND: Cardiac allograft vasculopathy after heart transplantation leads to an accelerated form of atherosclerosis with marked and often diffuse vessel wall changes that limit long-term survival. Previous studies showed contradictory results relating vessel wall changes to endothelial vasodilator response.
METHODS: A total of 30 cardiac transplant recipients were studied 3, 12, and 24 months after heart transplantation. Coronary angiography was performed at rest, during supine bicycle ergometry, and after 1.6 mg sublingual nitroglycerin. Coronary cross-sectional area (biplane coronary angiography) and coronary artery wall changes (intravascular ultrasound) were assessed and extent of intimal changes correlated to vasodilator responses to nitroglycerine and bicycle ergometry.
RESULTS: Intravascular ultrasound showed significant intimal thickening in 43, 64, and 58% of patients at 3, 12, and 24 months. Intimal thickening 3 months after transplantation was related to donor age (r=0.70, P<0.01) but did not predict progression of disease that manifested itself angiographically as a decrease in coronary cross-sectional area at 12 and 24 months (P<0.005) and significant coronary stenosis in 12% of patients after 24 months. Endothelium-independent vasodilatation after nitroglycerin (33+/-15, 44+/-20, and 43+/-24%) was normal. Endothelium-dependent, flow-induced vasodilatation during exercise was decreased (14+/-11, 18+/-14, and 16+/-17%) but did not correlate to intimal changes assessed by ultrasound.
CONCLUSIONS: The study confirms the high incidence of intimal thickening after heart transplantation as assessed by intravascular ultrasound. Impaired exercise-induced vasodilatation suggests diminished bioavailability of endothelium-derived nitric oxide to physiological stimulation but the lack of relationship between coronary wall changes and this functional impairment suggests intermittent and presumably reversible endothelial injury in graft atherosclerosis.

Entities:  

Mesh:

Year:  2000        PMID: 10755538     DOI: 10.1097/00007890-200003150-00030

Source DB:  PubMed          Journal:  Transplantation        ISSN: 0041-1337            Impact factor:   4.939


  10 in total

1.  Levels of circulating CXCR4-positive cells are decreased and negatively correlated with risk factors in cardiac transplant recipients.

Authors:  Colin Gerard Egan; Francesca Caporali; Pier Leopoldo Capecchi; Pietro Enea Lazzerini; Franco Laghi Pasini; Vincenzo Sorrentino
Journal:  Heart Vessels       Date:  2010-10-30       Impact factor: 2.037

2.  Magnetic Field Changes Macrophage Phenotype.

Authors:  Jarek Wosik; Wei Chen; Kuang Qin; Rafik M Ghobrial; Jacek Z Kubiak; Malgorzata Kloc
Journal:  Biophys J       Date:  2018-04-24       Impact factor: 4.033

3.  Upregulation of CD40 expression on endothelial cells infected with human cytomegalovirus.

Authors:  Tim Maisch; Barbara Kropff; Christian Sinzger; Michael Mach
Journal:  J Virol       Date:  2002-12       Impact factor: 5.103

4.  Prediction of long-term (> 10 year) cardiovascular outcomes in heart transplant recipients: Value of stress technetium-99m tetrofosmin myocardial perfusion imaging.

Authors:  Jesse F Veenis; Hendrik J Boiten; Jan C van den Berge; Kadir Caliskan; Alex P W M Maat; Roelf Valkema; Alina A Constantinescu; Olivier C Manintveld; Felix Zijlstra; Ron T van Domburg; Arend F L Schinkel
Journal:  J Nucl Cardiol       Date:  2017-11-07       Impact factor: 5.952

5.  Progenitor cells from the explanted heart generate immunocompatible myocardium within the transplanted donor heart.

Authors:  David A D'Alessandro; Jan Kajstura; Toru Hosoda; Alessandro Gatti; Ricardo Bello; Federico Mosna; Silvana Bardelli; Hanqiao Zheng; Domenico D'Amario; M Elena Padin-Iruegas; Adriana Bastos Carvalho; Marcello Rota; Michael O Zembala; David Stern; Ornella Rimoldi; Konrad Urbanek; Robert E Michler; Annarosa Leri; Piero Anversa
Journal:  Circ Res       Date:  2009-10-08       Impact factor: 17.367

6.  A reproducible mouse model of chronic allograft nephropathy with vasculopathy.

Authors:  Abolfazl Zarjou; Lingling Guo; Paul W Sanders; Roslyn B Mannon; Anupam Agarwal; James F George
Journal:  Kidney Int       Date:  2012-08-08       Impact factor: 10.612

7.  A Small Molecule β2 Integrin Agonist Improves Chronic Kidney Allograft Survival by Reducing Leukocyte Recruitment and Accompanying Vasculopathy.

Authors:  Samia Q Khan; Lingling Guo; David J Cimbaluk; Hatem Elshabrawy; Mohd Hafeez Faridi; Meenakshi Jolly; James F George; Anupam Agarwal; Vineet Gupta
Journal:  Front Med (Lausanne)       Date:  2014-11-12

Review 8.  Macrophage Proinflammatory Responses to Microorganisms and Transplanted Organs.

Authors:  Malgorzata Kloc; Ahmed Uosef; Jacek Z Kubiak; Rafik M Ghobrial
Journal:  Int J Mol Sci       Date:  2020-12-18       Impact factor: 5.923

9.  HMG-CoA reductase inhibition aborts functional differentiation and triggers apoptosis in cultured primary human monocytes: a potential mechanism of statin-mediated vasculoprotection.

Authors:  Joannis E Vamvakopoulos; Colin Green
Journal:  BMC Cardiovasc Disord       Date:  2003-07-19       Impact factor: 2.298

10.  Chronic allograft rejection: A significant hurdle to transplant success.

Authors:  Malgorzata Kloc; Rafik M Ghobrial
Journal:  Burns Trauma       Date:  2014-01-26
  10 in total

北京卡尤迪生物科技股份有限公司 © 2022-2023.