Literature DB >> 10711696

Kinetics of apoptotic cells in experimental autoimmune uveoretinitis.

J Sueda1, N Hikita, M Mochizuki, A Jimi, M Kojiro.   

Abstract

PURPOSE: To investigate the role of apoptosis in immunopathogenic mechanisms of experimental autoimmune uveoretinitis (EAU), the kinetics of apoptotic cells and expression of Fas and Fas ligand (FasL) in the eye with EAU were studied.
METHODS: Male inbred Lewis rats were immunized with S-antigen (40 microg/rat), and eyes were examined to detect apoptotic cells on days 1, 4, 8, and 10 post-immunization and days 0, 2, 4, 6, and 8 after the onset of EAU. The clinical and pathologic scores were used for estimating EAU. Apoptotic cells were analyzed by TdT-mediated dUTP nick-end labeling, electron microscopic and immunohistologic examinations, and agarose gel electrophoresis. The anti-rat Fas and anti-rat FasL antibodies were used to examine the expression of Fas and FasL.
RESULTS: Apoptotic cells were detected in the infiltrating cells in the aqueous humor, the vitreous body, the iris-ciliary body, and the retina. Apoptotic cells were observed as early as the day of EAU onset and reached a peak on day 2 after the disease onset. Fas and FasL were expressed on the infiltrating cells in the aqueous humor and the vitreous. FasL was expressed on resident cells of the ciliary body. The kinetics of the expression of FasL corresponded with the kinetics of apoptotic cells.
CONCLUSIONS: Fas-FasL-mediated apoptosis is considered to occur in the eye with EAU and plays a role in the immunopathogenic mechanisms to eliminate ocular infiltrating cells, thereby down-regulating the inflammatory processes.

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Year:  2000        PMID: 10711696

Source DB:  PubMed          Journal:  Invest Ophthalmol Vis Sci        ISSN: 0146-0404            Impact factor:   4.799


  6 in total

1.  Nitric oxide mediates apoptosis through formation of peroxynitrite and Fas/Fas-ligand interactions in experimental autoimmune uveitis.

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2.  Retinal pigment epithelial cells phagocytosis of T lymphocytes: possible implication in the immune privilege of the eye.

Authors:  F Willermain; L Caspers-Velu; B Nowak; P Stordeur; R Mosselmans; I Salmon; T Velu; C Bruyns
Journal:  Br J Ophthalmol       Date:  2002-12       Impact factor: 4.638

3.  Unlike Th1, Th17 cells mediate sustained autoimmune inflammation and are highly resistant to restimulation-induced cell death.

Authors:  Guangpu Shi; Madhu Ramaswamy; Barbara P Vistica; Catherine A Cox; Cuiyan Tan; Eric F Wawrousek; Richard M Siegel; Igal Gery
Journal:  J Immunol       Date:  2009-11-04       Impact factor: 5.422

4.  Apoptotic mechanisms within the retina in Staphylococcus epidermidis experimental endophthalmitis.

Authors:  Nikolaos M Pharmakakis; Ioannis K Petropoulos; Constantine D Georgakopoulos; Chrysoula V Vantzou; Evangelos D Anastassiou; Anestis Mavropoulos; Vassiliki Zolota
Journal:  Graefes Arch Clin Exp Ophthalmol       Date:  2008-11-28       Impact factor: 3.117

Review 5.  ER stress and apoptosis: a new mechanism for retinal cell death.

Authors:  Guangjun Jing; Joshua J Wang; Sarah X Zhang
Journal:  Exp Diabetes Res       Date:  2011-12-14

6.  Hypoxia inducible factors are dispensable for myeloid cell migration into the inflamed mouse eye.

Authors:  Peter J Gardner; Sidath E Liyanage; Enrico Cristante; Robert D Sampson; Andrew D Dick; Robin R Ali; James W Bainbridge
Journal:  Sci Rep       Date:  2017-01-23       Impact factor: 4.379

  6 in total

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